“…Briefly, a molecular pathway has been described in which a peptide derived from INFLORESCENCE DEFICIENT IN ABSCISSION (IDA) is thought to trigger a pair of redundant receptor-like protein kinases, HAESA (HAE) and HAESA-LIKE2 (HSL2). A mitogenactivated protein kinase (MAPK) pathway, consisting of MAPK KINASE4 (MKK4)/MKK5 and MAPK3 (MPK3)/MPK6, positioned genetically downstream of HAE/HSL2, phosphorylates a MADS-domain transcription factor, AGAMOUS-LIKE15 (AGL15), which results in a transcriptional increase of HAE, thereby creating a positive feedback loop Stenvik et al, 2008;Patharkar and Walker, 2015). NEVERSHED (NEV), an ADP-ribosylation factorGTPase-activating protein, is thought to traffic HAE/HSL2 and other receptor-like protein kinases to the correct subcellular location and is also required for abscission (Liljegren et al, 2009;Burr et al, 2011;Liljegren, 2012).…”