Fibromuscular disease of carotid arteries: long term results of graduated internal dilatation.

Starr, David S.; Lawrie, Gerald M.; Morris, George C.

doi:10.1161/01.str.12.2.196

Cited by 47 publications

(10 citation statements)

References 16 publications

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“…Contrary to this specific hypothesis, however, is the fact that presence of fibromuscular dysplasia in areas with dolichoarteriopathies is very rare. [28][29][30] …”

Section: Possible Embryological Mechanismsmentioning

confidence: 99%

Are Kinking and Coiling of Carotid Artery Congenital or Acquired?

et al. 2009

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Dolichoarteriopathies consist of tortuosity, kinking, or coiling of the extracranial carotid arteries. Some authors consider these alterations a consequence of atherosclerotic vessel remodeling, while others ascribe them to anatomical variations of embryological origin. The objective was to establish whether carotid dolichoarteriopathies belonged to a congenital origin or to acquired conditions. Color Doppler ultrasonography of neck vessels was performed in 885 participants, whose age ranged from 1-day-old infants to 90-year-old adults. Prevalence of kinking and coiling was evaluated, and it was related to the presence of cardiovascular risk factors. Prevalence of either kinking or coil of carotid arteries showed no increase with age, as it was comparable across all ages; furthermore, frequency of these alterations showed no relationship to cardiovascular risk factors nor to the presence of atheromatous plaques. These findings suggest that carotid dolichoarteriopathies are a result of alterations in embryological development rather than vascular remodeling secondary to aging and/or atherosclerosis.

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“…Contrary to this specific hypothesis, however, is the fact that presence of fibromuscular dysplasia in areas with dolichoarteriopathies is very rare. [28][29][30] …”

Section: Possible Embryological Mechanismsmentioning

confidence: 99%

Are Kinking and Coiling of Carotid Artery Congenital or Acquired?

et al. 2009

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“…The surgical technique depended on the type of lesion and its location, but the most widely used procedure was graduated intraluminal dilatation alone [68,69,75,76] or in combination with other procedures, such as resection of the diseased segment and primary anastomosis, autogenous saphenous vein grafting, aneurysm resection, and carotid endarterectomy [69,77,78]. Operative balloon angioplasty has also been used [79], and polytetrafluoroethylenecovered endografts have been deployed through a transverse incision in the neck and open puncture of the common carotid artery [80].…”

Section: Surgerymentioning

confidence: 99%

Fibromuscular dysplasia

Slovut¹,

Olin

2005

Curr Treat Options Cardio Med

193

100

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The most common clinical manifestations of fibromuscular dysplasia (FMD) are hypertension due to renal artery involvement and transient ischemic attack or stroke due to carotid or vertebral artery involvement. Patients with renal artery FMD and hypertension should undergo primary angioplasty with the goal of curing the hypertension. If the blood pressure fails to normalize following angioplasty, the physician should institute antihypertensive medications according to the recommendations of the Joint National Committee on the Prevention, Detection, and Treatment of High Blood Pressure VII. In patients with cerebrovascular FMD, antiplatelet agents represent the cornerstone of therapy. Percutaneous angioplasty has emerged as the preferred treatment for symptomatic cerebrovascular FMD.

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“…It also plays a role as alternative treatment strategy for extracranial artery stenosis [2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13]. PTA for the treatment of severe symptomatic carotid stenosis instead of carotid endarterectomy is controversely discussed [14, 15, 16, 17, 18].…”

Section: Introductionmentioning

confidence: 99%

Clinical and Ultrasonic Long-Term Results of Percutaneous Transluminal Carotid Angioplasty

Schöser

Becker²,

Eckert³

et al. 1998

Cerebrovasc Dis

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Experience of the long-term outcome of patients treated with carotid balloon angioplasty is limited. Therefore, we prospectively analyzed the ultrasonic and clinical features of 29 patients with complete follow-up data beyond 24 months, evaluated from 1989 through 1996 from our carotid angioplasty cohort of 106 patients. Mean follow-up time was 33 months. For up to 78 months postangioplasty, 23 patients with 24 angioplasties (77%) had no further neurological sequelae. Single ipsilateral amaurosis fugax or TIA events occurred in 3 patients. Recurrent ipsilateral amaurosis fugax or TIA events were noted twice in 2 patients. No patient suffered an ipsilateral stroke. Fifteen angioplasties (50%) remained with normal ultrasound (stenosis <50%), mild restenosis (50–70%) occurred in 12 angioplasties (40%), and severe restenosis (>70%) in 3 angioplasties (10%). Only in 2 of 15 patients clinical complications were related to the occurrence of ipsilateral restenosis above 50%. Until now, rigorous and careful evaluation of patients and clinical and ultrasonic follow-up have been essential for the estimation of the long-term efficacy of carotid angioplasty. It should be noted that carotid angioplasty is a new technique in evolution, with a high potential improving the technical results.

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Fibromuscular disease of carotid arteries: long term results of graduated internal dilatation.

Cited by 47 publications

References 16 publications

Are Kinking and Coiling of Carotid Artery Congenital or Acquired?

Are Kinking and Coiling of Carotid Artery Congenital or Acquired?

Fibromuscular dysplasia

Clinical and Ultrasonic Long-Term Results of Percutaneous Transluminal Carotid Angioplasty

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