Fetal Nutrition

Influence of the nutritional deficit of foetal survival and growth and plasma metabolites in rabbit does

“…In the rabbit, the last days of pregnancy correspond to the phase of maximal growth of the foetuses (Hudson and Hull, 1975), whereas the appetite of the doe decreases. The maternal tissue's spare glucose which is preferentially used by pregnant uterus (Hauguel et al, 1987;Battaglia and Meschia, 1988) (Prunier et al, 1993a(Prunier et al, , 1993b (Prunier et al, 1993a) and this could be due to a decrease in amino-acid catabolism, in relation to the lower amount of protein absorbed .…”

“…This hypothesis is also supported by the observation of lower birth weights in both the rabbit (Lebas, 1975) and other species (sow: Henry and Étienne, 1978; ewe: Robinson, 1990) Lebas, 1985; rat: Abel, 1990), whereas others observed no effect (rabbit: Lebas, 1975;pig: Henry and Étienne, 1978). (Battaglia and Meschia, 1988). Therefore, the concentrations of these metabolites were measured in plasma taken from does at 2 stages of gestation (d 17 and 28, experiment 2).…”

Blood urea nitrogen and serum bicarbonate in extremely low birth weight infants receiving higher protein intake in the first week after birth

“…Increased amino-acid intake is thought to result in increased urea and ammonia production. 9,19,20 However, as noted by Thureen et al, 4 'an elevated urea concentration in the ELBW infant may reflect an acceptable generation of an acceptable metabolic by-product and not protein intolerance.' A number of studies have disputed the association of hyperammonemia and metabolic acidosis with the amount of amino-acid intake in the early neonatal period.…”

Early postnatal nutritional requirements of the very preterm infant based on a presentation at the NICHD-AAP workshop on research in neonatology

“…12,13 Thus, the very preterm infant requires a large and continuous supply of glucose for energy metabolism. Because glycogen content is relatively limited in the very preterm infant, unless glucose is supplied directly, glucose deficiency and hypoglycemia commonly develop in these infants.…”