Feline Cardiology

Côté, Étienne; MacDonald, Kristin A.; Meurs, Kathryn M.; Sleeper, Meg M.

doi:10.1002/9781118785782

Cited by 59 publications

(51 citation statements)

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“…The same might be hypothesised for cats with HCM that show the 'burnt 4 However, no burnt out morphology was identified in our case series. The protective effect of asymmetrical IVS hypertrophy has also been related to SAM pathogenesis and to the association of a heart murmur, thus providing clinical tools to perform early diagnosis.…”

Section: Discussionsupporting

confidence: 81%

“…Arterial thromboembolism is therefore a marker of severe cardiovascular impairment. 4,26 Limitations of this study were mainly related to its retrospective nature. No systematic treatment protocols were performed, some clinical (T4 measurement) and echocardiographic data (ie, transmitral pulsed wave Doppler pattern) were not systematically assessed in our archive.…”

Section: Discussionmentioning

confidence: 98%

“…1,2 Hypertrophic cardiomyopathy (HCM) is the most common feline CM, and is characterised by a hypertrophied (>6 mm in diastole), non-dilated left ventricle (LV) in the absence of another systemic or cardiac disease able to mimic similar wall thickening. [1][2][3][4][5] HCM is characterised by a wide phenotypic spectrum, with diffuse hypertrophy (symmetrical and concentric hypertrophy of the LV), or asymmetric hypertrophy involving the interventricular septum (IVS) or the left ventricular free wall (LVFW) or isolated segments in the LV, including the papillary muscle. [4][5][6][7][8] The presence of a documented systemic illness that is responsible for LV wall thickening identifies a secondary CM.…”

Section: Introductionmentioning

confidence: 99%

“…[1][2][3][4][5] HCM is characterised by a wide phenotypic spectrum, with diffuse hypertrophy (symmetrical and concentric hypertrophy of the LV), or asymmetric hypertrophy involving the interventricular septum (IVS) or the left ventricular free wall (LVFW) or isolated segments in the LV, including the papillary muscle. [4][5][6][7][8] The presence of a documented systemic illness that is responsible for LV wall thickening identifies a secondary CM. 2,[9][10][11] Little is known about the prevalence and the consequences of cardiac abnormalities in cats affected by hyperthyroidism and systemic hypertension, and no study has investigated the outcome of these patients with a particular focus on the cardiovascular system.…”

Section: Introductionmentioning

confidence: 99%

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Survival in cats with primary and secondary cardiomyopathies

Spalla

Locatelli

Riscazzi

et al. 2015

Journal of Feline Medicine and Surgery

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Primary CMs in cats share some common features (ie, LA dimension and hypercoagulable state) linked to feline cardiovascular physiology, which influence survival greatly in end-stage CM. The presence of clinical signs has to be regarded as a marker of disease severity, regardless of the underlying CM. Secondary CMs are more benign conditions, but if the primary disease is not properly managed, the prognosis might also be poor in this group of patients.

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Section: Discussionsupporting

confidence: 81%

Section: Discussionmentioning

confidence: 98%

Section: Introductionmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

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Survival in cats with primary and secondary cardiomyopathies

Spalla

Locatelli

Riscazzi

et al. 2015

Journal of Feline Medicine and Surgery

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“…Ventricular septal defect (VSD) is among the most common feline congenital heart malformations (1,2). In cats, restrictive VSDs do not require treatment (3), but non-restrictive VSDs receive either palliative surgery or medical treatment to reduce pulmonary hypertension and excessive pulmonary blood flow.…”

Section: Introductionmentioning

confidence: 99%

Hybrid Surgical Approach Using Amplatzer Occluder for Treatment of VSD in a Cat

Uemura

Tanaka

2017

Macedonian Veterinary Review

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Ventricular septal defect (VSD) is among the most common feline congenital heart malformations. Although usually treated by pulmonary artery banding or pharmacotherapy, neither method is curative. Curative procedures have been performed in humans. Treatment of VSD is usually not required in dogs and cats, but is necessary in cases of non-restrictive VSD. Dogs with non-restrictive VSD are treated either using surgical correction under open-heart surgery, or percutaneous insertion of an occluder under interventional radiation. In our experience, neither method alone is appropriate for treating ventricular septal defect in cats with non-restrictive VSDs. We have applied a hybrid surgical method in a 13-month-old, female Maine Coon cat weighing 3.5 kg. A catheter was inserted and an occluder placed directly at the defect through thoracotomy. This method is less invasive than open-heart surgery and requires no special equipment, using direct puncture of the heart to minimize the distance to the defect and improving maneuverability. By combining the advantages of both surgical defect closure and occlusion guided by interventional ultrasound, treatment that could not be performed with either method alone became possible. In this case, the VSD could not be embolized, but some important points were suggested regarding surgical treatment of VSD in cats. One is that a sizing balloon catheter may be the most accurate method to measure VSD in cats. Amplatzer occlusion of a large non-restrictive VSD in a cat is currently not an option, but the described hybrid technique may still be an option for smaller non-restrictive VSDs. However, criteria and adjustments must be considered carefully.

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Acquired Heart Disease

Holland

2020

Feline Diagnostic Imaging

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Feline Cardiology

Cited by 59 publications

References 0 publications

Survival in cats with primary and secondary cardiomyopathies

Survival in cats with primary and secondary cardiomyopathies

Hybrid Surgical Approach Using Amplatzer Occluder for Treatment of VSD in a Cat

Acquired Heart Disease

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