Fanconi Syndrome Following Homotransplantation

Vertuno, LCDR Leonard L.

doi:10.1001/archinte.1974.00320140140016

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Pathophysiology Of Hyperuricemia In Renal Transplantation1

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1978

2004

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Cited by 15 publications

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“…This argues against any specific impairment in uric acid excretion inherent to transplantation itself [15]. In some patients, hypouricemia and hyperuricosuria were reported and attributed to the uricosuric effect of steroids [16] or to proximal tubular dysfunction [17].…”

Section: Pathophysiology Of Hyperuricemia In Renal Transplantationmentioning

confidence: 99%

Hyperuricemia in Kidney Transplantation

Perico

Codreanu

Caruso

et al. 2004

Contributions to Nephrology

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Hyperuricemia is a common problem among renal transplant recipients. Its prevalence is clearly attributable to cyclosporine (CsA) use, although individual patients may have other risk factors as well. CsA lowers the urinary clearance of uric acid. The specific mechanism for this is unknown, but may involve alteration in tubular transport. Hyperuricemia may add on to several other factors in contributing to progressive deterioration of graft function and ultimately graft loss. The therapy of hyperuricemia may be particularly challenging in transplant patients.

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Section: Pathophysiology Of Hyperuricemia In Renal Transplantationmentioning

confidence: 99%