Famili�rer Faktor X-Mangel

Oehme, J; Schwick, G.; Schultze, H. E.

doi:10.1007/bf01488872

Cited by 8 publications

(2 citation statements)

References 12 publications

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“…Specifically, from clinical and coagulation studies, it is concluded that the effects on factor X deficiency caused by pregnancy or norethynodrel are due to excessive progesterone or norethynodrel, which produces increased factor X production, rather than acting as a substitute for factor X. Nonetheless, controversy still remains with regard to this point (Johnson 1957 ;Oehme et al 1958 ;McGovern et al 1961). …”

Section: Therapymentioning

confidence: 99%

Congenital factor X deficiency in Japan.

Mori

Sakai

Nakano

et al. 1981

Tohoku J. Exp. Med.

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Section: Therapymentioning

confidence: 99%

Congenital factor X deficiency in Japan.

Mori

Sakai

Nakano

et al. 1981

Tohoku J. Exp. Med.

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“…To date only about 25 proven or probable cases have been reported in the literature [2-4, 6-8, 14, 19,21,23,[25][26][27][28][29][30][31]. The reasons for such rarity are unknown and seem really striking if one takes into ac count the fact that congenital factor VII deficiency, a condition which re sembles factor X deficiency, is at least 4 times more frequent [16], Recently several patients with a coagulation disorder due to the pres ence of an abnormal factor X have been described [13,14,15,17].…”

mentioning

confidence: 99%

Classical Factor X Deficiency

Girolami¹,

Cóser²,

Brunetti³

et al. 1975

Acta Haematol

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A case of classical factor X deficiency is reported. The propositus is a 28-year-old male who presented easy bruising, epistaxis, hematomas, hematuria and occasional hemartrosis since early childhood. The severely prolonged prothrombin time was corrected by normal serum but not by adsorbed normal plasma. The abnormality was not corrected by the plasma of a patient with factor X deficiency, but by the plasma of patients with factor II or VII deficiencies. Partial thromboplastin time, prothrombin consumption and the thromboplastin generation test were abnormal. The thromboelastogram showed a prolonged ‘K’ and ‘r’ together with a normal ‘ma’. Factor X was very low (<1%). Platelet tests were normal. No factor X band or precipitates were seen on electroimmunoassay and on the cross-over electrophoresis. The non-consanguineous parents and several other members of the family were found to be heterozygotes.

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Blutgerinnung, Thrombocyten und Blutgefäße

Künzer

1950

Die Physiologische Entwicklung Des Kindes

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Famili�rer Faktor X-Mangel

Cited by 8 publications

References 12 publications

Congenital factor X deficiency in Japan.

Congenital factor X deficiency in Japan.

Classical Factor X Deficiency

Blutgerinnung, Thrombocyten und Blutgefäße

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