The recognition of vitamin Bg as a new member of the vitamin Bcomplex is credited to Gyorgy (1935a, 1935b) who reported a "rat antipellagra factor" which he named vitamin Bg. These reports have been followed by extensive research and reports regarding the requirements, importance, and interrelationships of this vitamin. Most of the reports concern research with humans, or laboratory animals such as rats and mice, while reports on domestic fowl are more limited. Requirements and Deficiency Symptoms in Domestic Fowl Jukes (1939) and Hegsted ej: ajL. (1939) were two of the first to show that chicks needed a dietary source of vitamin Bg. These reports were followed shortly by those of Hegsted ej: al. (1940), Hogan et al. (1941) , Lepkovsky and Kratzer (1942), and Briggs et jil. (1942). These researchers generally reported vitamin Bg deficiency symptoms in chicks as inefficient food utilization, reduced food consumption, reduced growth, and death. Some groups observed spasmodic convulsions preceding death. Daghir and Balloun (1963) also observed ruffled feathers and wing feather follicle hemorrhages in vitamin Bg-deficient chicks. Miller (1963) observed an abnormal incidence of pendulous crop in pyridoxine-deficient chicks. Hogan jet al. (1941) reported the chick's vitamin Bg requirement to be between 300 and 500 meg of vitamin Bg per 100 g of ration. Briggs jet al. (1942) reported the requirement of chicks as 275 to 300 meg of vitamin Bg per 100 g of ration. Research by Lucas et al. (1946) indicated that vitamin Be requirements may vary with the breed of chicken. Daghir and