Facial paralysis due to a benign parotid tumour

Whillis, D.; Goepel, J R; Shorthouse, A. J.

doi:10.1002/bjs.1800760130

Cited by 9 publications

(2 citation statements)

References 13 publications

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“…Although several studies analyzing large series of patients with parotid tumors reported the occurrence of facial palsy only in patients with malignancy, [4][5][6] facial palsy due to benign parotid gland tumors has also been reported. A comprehensive review of the literature revealed 17 such cases [7][8][9][10][11][12][13][14][15][16][17][18][19][20][21][22] (►Table 1). The present case differs from the ones already discussed in the literature in two aspects.…”

Section: Discussionmentioning

confidence: 99%

Facial Nerve Paralysis due to a Pleomorphic Adenoma with the Imaging Characteristics of a Facial Nerve Schwannoma

et al. 2014

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Background Facial nerve paralysis in a patient with a salivary gland mass usually denotes malignancy. However, facial paralysis can also be caused by benign salivary gland tumors. Methods We present a case of facial nerve paralysis due to a benign salivary gland tumor that had the imaging characteristics of an intraparotid facial nerve schwannoma. Results The patient presented to our clinic 4 years after the onset of facial nerve paralysis initially diagnosed as Bell palsy. Computed tomography demonstrated filling and erosion of the stylomastoid foramen with a mass on the facial nerve. Postoperative histopathology showed the presence of a pleomorphic adenoma. Facial paralysis was thought to be caused by extrinsic nerve compression. Conclusions This case illustrates the difficulty of accurate preoperative diagnosis of a parotid gland mass and reinforces the concept that facial nerve paralysis in the context of salivary gland tumors may not always indicate malignancy.

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Section: Discussionmentioning

confidence: 99%

Facial Nerve Paralysis due to a Pleomorphic Adenoma with the Imaging Characteristics of a Facial Nerve Schwannoma

et al. 2014

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“…The tenacity of the extratemporal portion of the nerve to withstand significant compression is borne out in a review of the literature describing benign tumors of the parotid gland that caused facial dysfunction. Twenty‐four cases reported in the literature were analyzed 60‐79 . Six patients had tumorous extension into the stylomastoid foramen, where the compressive effects can easily explain the onset of facial palsy.…”

Section: Facial Dysfunction With Pressurementioning

confidence: 99%

Plasticity of the Extracranial Facial Nerve

Anand

1997

The Laryngoscope

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Although the expansion properties of peripheral nerves have been a matter of considerable study in recent years, investigations of the plasticity of cranial nerves, including the facial nerve, have been lacking. Clinicians, however, have long recognized the tenacity of facial nerve function in patients with slow-growing benign tumors that enormously distort the nerve. An experimental study was designed to assess whether tissue expansion techniques can be applied to the extracranial portion of the facial nerves of cats. In eight cats the frontozygomatic branch of the facial nerve was expanded by stages in seven sessions over a period of 40 days. The length of the nerve increased an average of 95% without significantly impairing nerve function. Pressure changes in the expander averaged 75 mm Hg during each stage of expansion. Electroneurography was performed after each injection of the expander. Statistical analysis of these data did not show consistent evidence of demyelination or denervation, and all but one cat exhibited a normal blink reflex and had normal electromyographic findings at the end of the experiment. Histologic examination of the expanded nerves, however, did show inflammatory changes, intraneural edema, and occasional demyelination.

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