Eyelid Dermatitis: An Evaluation of 447 Patients

Ayala, Fabio; Fabbrocini, Gabriella; Bacchilega, Roberto; Berardesca, Enzo; Caraffini, Stefano; Corazza, Monica; Flori, Maria Laura; Francalanci, Simona; Guarrera, Marcella; Lisi, Paolo; Santucci, B.; Schena, Donatella; Suppa, Francesco; Valsecchi, R.; Vincenzi, Colombina; Balato, Nicola

doi:10.2310/6620.2003.38359

Cited by 18 publications

(36 citation statements)

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“…[1,2,13] The eyelids are particularly predisposed to ABCD because they are exposed and materials may lodge and get deposited on the upper eyelids. Hence there is also ABCD, caused by things such as pollen, dust mites, animal dander, and chemicals suspended in the air.…”

Section: Discussionmentioning

confidence: 99%

Spectrum of periorbital dermatoses in South Indian population

Besra

Jaisankar

Thappa

et al. 2013

Indian J Dermatol Venereol Leprol

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Section: Discussionmentioning

confidence: 99%

Spectrum of periorbital dermatoses in South Indian population

Besra

Jaisankar

Thappa

et al. 2013

Indian J Dermatol Venereol Leprol

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“…Lichenification, scaling and fissuring are features of more chronic disease. The incidence of ACD as a cause of eyelid dermatitis varies from 29 to 77% of patients reported,1–3 5 7 8 10 and has been found to be the most likely cause if all four eyelids are involved 10. It is more common in middle-aged patients with less pigmented skin.…”

Section: Discussionmentioning

confidence: 99%

“…Amin et al 11 reported 85.4% of their patients with ACD as Caucasian in origin with the greatest prevalence in the 41–70-year-old age range. Females are most frequently affected (61.8–90% of patients) because of the use of cosmetics 1 2 5 7 8 10…”

Section: Discussionmentioning

confidence: 99%

Allergic contact dermatitis: a case series and review for the ophthalmologist

Morris

Barlow

Selva

et al. 2010

British Journal of Ophthalmology

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Allergic contact dermatitis: a case series and review for the ophthalmologist ABSTRACTEyelid dermatitis is most commonly caused by an allergenic response, potentially from exposure at another site, rather than from local toxicity. Yet allergic contact dermatitis is a diagnosis often missed by ophthalmologists. The authors review the literature and detail their experience relating to the causes, clinical features and management of this condition. 14 patients over a 2-year period that were referred to the oculoplastic service for a further opinion were reviewed in a retrospective, non-comparative study.

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“…[15][16][17] Although EBA is a rare disease, we recommend that, in the absence of typical anamnestic or clinical stigmata of more frequent causes of eyelid dermatitis (such as allergic contact dermatitis, irritant contact dermatitis, or atopic dermatitis), or in the case of a therapy-resistant course, a careful histologic and immunologic examination is warranted to avoid delayed diagnosis of a potentially life-threatening autoimmune bullous disease. [15][16][17] Although EBA is a rare disease, we recommend that, in the absence of typical anamnestic or clinical stigmata of more frequent causes of eyelid dermatitis (such as allergic contact dermatitis, irritant contact dermatitis, or atopic dermatitis), or in the case of a therapy-resistant course, a careful histologic and immunologic examination is warranted to avoid delayed diagnosis of a potentially life-threatening autoimmune bullous disease.…”

Section: Discussionmentioning

confidence: 99%

Premonitory Epidermolysis Bullosa Acquisita Mimicking Eyelid Dermatitis

Kubisch

Diessenbacher

Schmidt

et al. 2010

American Journal of Clinical Dermatology

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We report a unique case of a 71-year-old female patient with epidermolysis bullosa acquisita. The patient initially presented with the clinical symptoms of bilateral eyelid dermatitis that occurred several months prior to the development of oral and pharyngeal erosions and blisters. While no contact allergy was found by patch testing, direct immunofluorescence microscopy demonstrated linear deposits of IgG at the basement membrane zone. By indirect immunofluorescence microscopy on human salt-split skin, IgG antibodies bound to the dermal side of the split. Immunoblot analysis showed predominant IgG4 reactivity of the patient's serum with the recombinant non-collagenous-1 domain of type VII collagen. Because of treatment resistance to systemic corticosteroids, dapsone, and colchicine, we initiated a combination treatment of protein A immunoapheresis and rituximab. With this treatment, complete remission was achieved within 4 months. Our case highlights that epidermolysis bullosa acquisita may initially mimic an eyelid dermatitis. Consequently, dermatologists should be aware of this rare differential diagnosis of eyelid dermatitis where a contact allergy or atopic dermatitis is absent.

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Eyelid Dermatitis: An Evaluation of 447 Patients

Cited by 18 publications

References 0 publications

Spectrum of periorbital dermatoses in South Indian population

Spectrum of periorbital dermatoses in South Indian population

Allergic contact dermatitis: a case series and review for the ophthalmologist

Premonitory Epidermolysis Bullosa Acquisita Mimicking Eyelid Dermatitis

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