Experience with renal failure during extracorporeal membrane oxygenation: Treatment with continuous hemofiltration

Sell, Linda L.; Cullen, Marc L.; Whittlesey, Grant C.; Lerner, Gary; Klein, Michael D.

doi:10.1016/s0022-3468(87)80107-0

Cited by 55 publications

(24 citation statements)

References 7 publications

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“…3,4 Diuretic therapy and renal replacement therapy are not uncommon in this setting, further complicating the fluid and electrolyte management of these patients. [5][6][7] Increases in atrial natriuretic hormone and plasmin renin activity have also been observed and quantified during ECMO. 2 Visser and de Jong 8 demonstrated that leakage from the membrane oxygenator was dependent on the type of hollowfiber membrane.…”

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confidence: 99%

Insensible Water Loss From the Medtronic Minimax Oxygenator: An In Vitro Study

et al. 2006

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The purposes of this study were to quantify the insensible water loss that occurs across the Medtronic Minimax oxygenator and to estimate the resultant rise in fluid sodium concentration.A Carmeda-coated extracorporeal membrane oxygenation circuit connected to a Medtronic Minimax Plus oxygenator was primed with normal saline and attached to a closed reservoir. The gas sweep was randomly assigned to one of three rates: 2, 5, or 10 LPM (liters per minute). Each sweep rate was run in triplicate. The sodium concentration of the circuit was assessed after 12 and 24 hours of each trial. At the end of each 24-hour run, the evaporative loss was calculated. The average insensible water losses were 6.9+/-0.4 ml/h, 16.6+/-1.5 ml/h, and 34.4+/-0.3 ml/h at gas sweep rates of 2, 5, and 10 LPM, respectively (p<0.0001). Daily evaporative water losses for the membrane can be estimated to be 82.7+/-2.2 ml for each 1 LPM of sweep gas flow for a normal saline pump flow of 300 ml/min. In a closed circuit, a faster sweep gas rate is associated with a more rapid rise in sodium concentration (p<0.0001).

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mentioning

confidence: 99%

Insensible Water Loss From the Medtronic Minimax Oxygenator: An In Vitro Study

et al. 2006

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“…Acute renal failure and the consequences of this organ dysfunction, such as hypervolemia, hyperkalemia, or azotemia, are indications for continuous hemofiltration placed in-line with the ECMO circuit (59).…”

Section: Complicationsmentioning

confidence: 99%

Pediatric Extracorporeal Membrane Oxygenation: An Introduction for Emergency Medicine Physicians

Gehrmann¹,

Hafner

Montgomery

et al. 2015

The Journal of Emergency Medicine

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“…For this reason, feeding with breastmilk is recommended for newborns with AKI. Significant elevations in serum phosphate represent a risk of development of extraskeletal calcifications of the heart, blood vessels, and kidneys in the newborn, especially when the calcium-phosphorus product exceeds 70 (23). Calcium carbonate may be used as a phosphate-binding agent in infants whose phosphorous intake exceeds excretion.…”

Section: Management Medical Managementmentioning

confidence: 99%

Neonatal Acute Kidney Injury

Zübarioğlu¹,

Bülbül²,

Uslu³

2013

JAREM

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Acute kidney injury (AKI) is characterized by a sudden deterioration in kidney function that results in the accumulation of nitrogenous waste products (e.g., urea) and alters the regulation of extracellular fluid volume, electrolytes, and acid-base homeostasis. Although the criteria for neonatal acute kidney injury have varied, a frequently used definition is a serum creatinine level of more than 1.5 mg/dL.The causes of neonatal acute kidney injury are multiple and can be divided into prerenal, renal, and postrenal categories. Prerenal azotemia is the most common type of acute kidney injury in the neonate and may account for up to 85% of all cases. There are currently no specific medical therapies to treat AKI. The basic approach in management of AKI should be planned according to the underlying etiology. To maximize the chance for survival, the clinician must support the cardiorespiratory system, maintain maximal nutrition, balance homeostasis, and manage the consequences of AKI. The prognosis for neonates with acute kidney injury is variable, and largely related to the infant's underlying medical condition, with mortality rates ranging from 14% to 73%. (JAREM 2013; 3: 53-9) Key Words: Neonatal, acute, kidney injury ÖZET Akut böbrek hasarı; böbrek fonksiyonlarında ani bozulma ile karakterizedir. Sonuç olarak nitrojen yıkım ürünlerinin birikimi, ekstrasellüler sıvı hacmi, elektrolit ve asid-baz dengesinin regülasyonunda değişiklikler olur. Neonatal akut böbrek hasarı kriterleri değişmekle beraber, sıklıkla serum kreatinin düzeyinin 1,5 mg/dL'yi geçmesi tanım olarak kabul edilebilir. Neonatal akut böbrek hasarının birçok nedeni vardır ve prerenal, renal ve postrenal olarak kategorilere ayrılabilir. Prerenal azotemi yenidoğan döneminde en sık görülen kategoridir ve tüm vakaların yaklaşık %85'i bu gruptandır. Akut böbrek hasarının halen günümüzde spesifik bir medikal tedavisi yoktur. Hastayı yönetirken temel yaklaşım altta yatan etiyolojiye göre planlanmalıdır. Hastanın yaşama şansını yükseltmek adına klinisyen, bebeğin kardiyopulmoner sistemini desteklemeli, maksimal beslenmeyi sağlamalı, homeostazı dengelemeli ve akut böbrek hasarının sonuçlarını yönetebilmelidir. Akut böbrek hasarı ile başvuran yenidoğanlarda prognoz değişkendir ve çoğunlukla bebeğin altta yatan medikal durumuyla ilişkilidir. Mortalite oranları %14 ile %73 arasında değişmektedir. (JAREM 2013; 3: 53-9) Anahtar Sözcükler: Yenidoğan, akut, böbrek hasarı

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Experience with renal failure during extracorporeal membrane oxygenation: Treatment with continuous hemofiltration

Cited by 55 publications

References 7 publications

Insensible Water Loss From the Medtronic Minimax Oxygenator: An In Vitro Study

Insensible Water Loss From the Medtronic Minimax Oxygenator: An In Vitro Study

Pediatric Extracorporeal Membrane Oxygenation: An Introduction for Emergency Medicine Physicians

Neonatal Acute Kidney Injury

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