Examination of central body fat deposition as a risk factor for loss-of-control eating

Berner, Laura A.; Mayer, Laurel; Sarwer, David B.; Lowe, Michael R.

doi:10.3945/ajcn.115.107128

Cited by 15 publications

(15 citation statements)

References 62 publications

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“…Eating behavior is formed under the influence of family, national, psychophysiological features, socio-cultural and ethical values, features of metabolism in the body and health. It depends on the interaction of the feeling of hunger and satiety, appetite, the ratio of motivational and emotional components [5][6][7][8].…”

Section: Introductionmentioning

confidence: 99%

Eating behavioral reactions of patients with chronic non-communicable diseases and its correction

Shalimova

Isayeva

Vovchenko

et al. 2020

МСЗ

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The peculiarities of the eating behavioral reactions of patients with chronic non-communicable diseases and the effect of group and individual therapy on them were evaluated. It was established, that with a tendency to positive changes in eating behavior, conducting both group and individual therapy for 1 year did not significantly change its indicators such as daily consumption of salt, vegetables and fruits, smoking and alcohol consumption, which confirms the complexity of the impact on the specified risk factor for the development and progression of chronic non-communicable diseases.

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Section: Introductionmentioning

confidence: 99%

Eating behavioral reactions of patients with chronic non-communicable diseases and its correction

Shalimova

Isayeva

Vovchenko

et al. 2020

МСЗ

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“…14 Additionally, how much of an individual's BMI is due to fat, and where this fat is located, might also be of importance, as these factors can influence body dissatisfaction and social stigma, with negative psychological consequences. 15 The aim of this study was to increase the understanding of the obesity-depression association by assessing the relationship between specific and biologically informative components of BMI (fat mass (stratified on limbs and trunk), non-fat mass (stratified on limbs and trunk) and depression via a MR study using results from large GWAS.…”

Section: Introductionmentioning

confidence: 99%

Investigating the Association Between Body Fat and Depression via Mendelian Randomization

Speed

Jefsen

Boerglum

et al. 2019

Preprint

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Obesity and depression are major public health concerns that are both associated with substantial morbidity and mortality. There is a considerable body of literature linking obesity to the development of depression. Recent studies using Mendelian Randomization indicate that this relationship is causal. Most studies of the obesity-depression association have used body mass index as a measure of obesity. Body mass index is defined as weight (measured in kilograms) divided by the square of height (meters) and therefore does not distinguish between the contributions of fat and non-fat to body weight. To better understand the obesity-depression association, we conduct a Mendelian Randomization study of the relationship between fat mass, non-fat mass, height, and depression, using genome-wide association study results from the UK Biobank (n=332,000) and the Psychiatric Genomics Consortium (n=480,000). Our findings suggest that both fat mass and height (short stature) are causal risk factors for depression, while non-fat mass is not. These results represent important new knowledge on the role of anthropometric measures in the etiology of depression. They also suggest that reducing fat mass will decrease the risk of depression, which lends further support to public health measures aimed at reducing the obesity epidemic.

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“…These two factors provide a deeper understanding of the pathophysiology of obesity and its potential metabolic cardiovascular or hepatic complications [2,4,5]. A small or even moderate weight loss significantly reduces the number of circulating inflammatory markers, regulates the profile of proinflammatory adipose tissue genes and the risks associated with obesity and other metabolic diseases [6].…”

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confidence: 99%

“…Increasing the percentage of VAT and imbalance of immunomodulatory proteins (adipokines) is considered to be the key link in the development of cardiovascular diseases (hypertension, atherosclerosis, myocardial infarction). A number of researchers are considering predicting the course of the cardiovascular diseases not the body mass index, namely the area or percentage of VAT [5,6,10]. The adipose tissue synthesizes angiotensin, which, under the influence of renin and angiotensin-converting enzyme, is converted into angiotensin II, which causes vasospasm and stimulates the synthesis of triglycerides [8,9].…”

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confidence: 99%

Metabolic role of visceral adipose tissue and main methods of its diagnosis at the present stage

Фадєєнко

Nikiforova

2020

МСЗ

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A review of the literature on the metabolic role of visceral adipose tissue and the main methods for its diagnosis is presented. Visceral adipose tissue is an active endocrine organ what secretes a number of biologically active substances. With an increase in the proportion of visceral adipose tissue, moderate inflammation is observed with a chronic systemic increase in the activity of adipokines. Adipokines carry out several immune or metabolic functions associated with inflammatory infiltration. Active substances such as leptin, adiponectin, resistin, etc., the source of which is visceral adipose tissue, have peripheral, central and local effects on the metabolism of glucose and lipids, glycolysis processes in the liver, etc. It is the activity of visceral adipose tissue that should be considered among the main pathophysiological development factors obesity and its potential metabolic cardiovascular and/or liver complications.

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Examination of central body fat deposition as a risk factor for loss-of-control eating

Abstract: These findings lend further support to the premise that increased central body fat deposition is associated with body image dissatisfaction and suggest that it may represent a risk and maintenance factor for LOC eating. This trial was registered at clinicaltrials.gov as NCT00456131.

Cited by 15 publications

References 62 publications

Eating behavioral reactions of patients with chronic non-communicable diseases and its correction

Eating behavioral reactions of patients with chronic non-communicable diseases and its correction

Investigating the Association Between Body Fat and Depression via Mendelian Randomization

Metabolic role of visceral adipose tissue and main methods of its diagnosis at the present stage

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