EVI1, BAALC and AME: Prevalence of the secondary mutations in chronic and accelerated phases of chronic myeloid leukemia patients from eastern India

Kuila, Nivedita; Sahoo, Devi Prasad; Kumari, Monika; Biswas, Sutapa; Patnaik, Rupali; Pattnayak, Naresh Chandra; Biswas, Ghanashyam; Chakraborty, Soumen

doi:10.1016/j.leukres.2008.07.018

Search citation statements

Order By: Relevance

Paper Sections

Select...

(Received 22 October 2008; Accepted 27 December 2008)1

Citation Types

Supporting

Mentioning

Contrasting

Year Published

2009

2021

Publication Types

Select...

Article5

Relationship

Self Cite2

Independent3

Authors

Journals

Cited by 5 publications

(1 citation statement)

References 5 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Although lot of our other patients progressed to advanced phase, however, we were not able to detect the BCR-ABL kinase domain mutation in them. This may be so as progression of the disease may involve other mechanisms or other secondary mutations [9].…”

Section: (Received 22 October 2008; Accepted 27 December 2008)mentioning

confidence: 99%

Presence of a newBCR-ABLkinase domain mutation, C330G in an imatinib naïve patient with chronic myeloid leukemia: very low prevalence ofBCR-ABLkinase domain mutations in patients with chronic myeloid leukemia from eastern India

Kuila¹,

Dash²,

Sahoo³

et al. 2009

Leukemia & Lymphoma

Self Cite

View full text Add to dashboard Cite

Section: (Received 22 October 2008; Accepted 27 December 2008)mentioning

confidence: 99%

Presence of a newBCR-ABLkinase domain mutation, C330G in an imatinib naïve patient with chronic myeloid leukemia: very low prevalence ofBCR-ABLkinase domain mutations in patients with chronic myeloid leukemia from eastern India

Kuila¹,

Dash²,

Sahoo³

et al. 2009

Leukemia & Lymphoma

Self Cite

View full text Add to dashboard Cite

Physical and functional interaction of the proto-oncogene EVI1 and tumor suppressor gene HIC1 deregulates Bcl-xL mediated block in apoptosis

Pradhan

Halder

Chakraborty

2014

The International Journal of Biochemistry & Cell Biology

View full text Add to dashboard Cite

EV/1-Positive Leukemias and Myelodysplastic Syndromes: Theoretical and Clinical Aspects (Literature Review)

Мамаев

Shakirova

Morozova

et al. 2021

Клиническая онкогематология

View full text Add to dashboard Cite

The present review provides the analysis of theoretical background and therapy of prognostically poorest EV/1-positive myeloid leukemias and myelodysplastic syndromes which is performed at the RM Gorbacheva Scientific Research Institute of Pediatric Oncology, Hematology and Transplantation. The focus is on the evidence of the dominating role of EV/1 gene in impaired epigenetic regulation of hematopoiesis and, thus, on the feasibility of allogeneic hematopoietic stem cell transplantation with hypomethylating agents and/ or trans-retinoic acid used for these diseases treatment.

show abstract

EVI1, BAALC and AME: Prevalence of the secondary mutations in chronic and accelerated phases of chronic myeloid leukemia patients from eastern India

Cited by 5 publications

References 5 publications

Presence of a newBCR-ABLkinase domain mutation, C330G in an imatinib naïve patient with chronic myeloid leukemia: very low prevalence ofBCR-ABLkinase domain mutations in patients with chronic myeloid leukemia from eastern India

Presence of a newBCR-ABLkinase domain mutation, C330G in an imatinib naïve patient with chronic myeloid leukemia: very low prevalence ofBCR-ABLkinase domain mutations in patients with chronic myeloid leukemia from eastern India

Physical and functional interaction of the proto-oncogene EVI1 and tumor suppressor gene HIC1 deregulates Bcl-xL mediated block in apoptosis

EV/1-Positive Leukemias and Myelodysplastic Syndromes: Theoretical and Clinical Aspects (Literature Review)

Contact Info

Product

Resources

About