1945
DOI: 10.1001/archinte.1945.00210330002001
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Etiology and Pathogenesis of Rheumatic Fever

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1945
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Cited by 77 publications
(10 citation statements)
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“…This incidence is comparable to the value of other observers (9,10 (Figure 2). The greatest rise ( 1150 units) is graphically shown in Figure 3.…”
Section: Resultssupporting
confidence: 89%
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“…This incidence is comparable to the value of other observers (9,10 (Figure 2). The greatest rise ( 1150 units) is graphically shown in Figure 3.…”
Section: Resultssupporting
confidence: 89%
“…More recently, Rantz, Boisvert, and Spink (10) have described a significant antistreptolysin response in 87.5 per cent of 342 patients with streptococcal sore throat. All three groups of workers (9,10,19) have also commented on the correlation of acute attacks of rheumatic fever and a rise in antisteptolysin titer. Similar rises have been noted in acute glomerulonephritis.…”
Section: Significance Of the Antistreptolysin Responsementioning
confidence: 99%
“…Epidemiological and clinical studies have clearly established that antecedent streptococcal infection is a prerequisite to rheumatic fever (92,165,205,206,248). Widespread involvement of connective tissue structures, including endocardium, heart valves, myocardium, pericardium, lung and pleura, tendons, joints, subcutaneous tissue, and finally brain function, is supported by the clinical picture of the disease, as well as by numerous pathological studies.…”
Section: Possible Role Of Immunological Mechanisms In the Diseasementioning
confidence: 99%
“…The fluid obtained from the joints in these cases is sterile. Many years ago, Swift (69,70) and others (182) established that infections of the upper respiratory tract with non-hcmolytic streptococci preceded by a variable time interval typical acute rheumatic fever. It is speculated that the antigen may be an auto-antigen activated by the streptococcus (183,184).…”
Section: Clinical Evidence Of Hypersensitivity As a Basismentioning
confidence: 99%