Estimating the Atmospheric Deposition of Polychlorinated Dibenzo-p-dioxins and Dibenzofurans from Soils

Spatial distribution of polychlorinated dibenzo-p-dioxins and polychlorinated dibenzofurans in soil around a municipal solid waste incinerator

“…Therefore, they readily accumulate in the environment, especially in organic carbon-rich media such as soil, sediment and biota (Meneses et al 2002;Polder et al 2008). Among these media, soil has been demonstrated to act as a conservative matrix for the atmospheric deposition of PCDD/Fs and to reflect long-term exposure (Brzuzy and Hites 1995). To date, many researchers have focused on the concentrations of PCDD/Fs in ambient soil around municipal solid waste incinerators (MSWIs) and their impact on the environment (Caserini et al 2004;Rovira et al 2010;Lorber et al 1998).…”

Contamination and Distribution of Polychlorinated Dibenzo-p-dioxins and Dibenzofurans (PCDD/F) in Agriculture Fields in Ya-Er Lake Area, China

“…Over the past several years, considerable interest has centered on environmental behavior of polychlorinated dibenzo-p-dioxins and dibenzofurans (PCDD/Fs) (Fletcher and McKay, 1993;Brzuzy and Hites, 1995). Among them, 2,3,7,8-TCDD is known as a human carcinogen and has been implicated as a potent endocrine disrupter for humans and other animals (Safe, 1990;IEH, 1995;Mocarelli et al, 1996).…”

Long-Term Toxic Impact of 2,3,7,8-Tetrachlorodibenzo-p-dioxin on the Reproduction, Sexual Differentiation, and Development of Different Life Stages of Gobiocypris rarus and Daphnia magna

“…TCDD is one of the most toxic man-made substances. It can be released into the environment as a trace product of various chemical and combustion processes (Fletcher and McKay, 1993;Brzuzy and Hites, 1995;Bonn, 1998). Because of its chemical stability, lipophilicity, and resistance to degradation, it is easy to bioaccumulate in aquatic species and it can cause acute, chronic, and subchronic toxicities, a!ecting reproduction and development (Safe, 1990;Brzuzy and Hites, 1995;IEH, 1995;Mocarelli et al, 1996;Bruckmeier et al, 1997).…”

“…It can be released into the environment as a trace product of various chemical and combustion processes (Fletcher and McKay, 1993;Brzuzy and Hites, 1995;Bonn, 1998). Because of its chemical stability, lipophilicity, and resistance to degradation, it is easy to bioaccumulate in aquatic species and it can cause acute, chronic, and subchronic toxicities, a!ecting reproduction and development (Safe, 1990;Brzuzy and Hites, 1995; IEH, 1995;Mocarelli et al, 1996;Bruckmeier et al, 1997). The toxic mechanism of TCDD is mediated through combining with the cellular Ah-receptor, after a series of synthesis of protein, the most sensitive biochemical response is considered to be the induction of 7-ethoxyresoru"n-O-deethylase (EROD) activity (Poland and Knutson, 1982;Landers and Brunce, 1991;Safe, 1994;Boening, 1998).…”