Esotropia Following Occlusion

Swan, Kenneth C.

doi:10.1001/archopht.1947.00890220457004

Cited by 46 publications

(20 citation statements)

References 6 publications

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“…Common to all 3 types is acute onset, concomitancy, a relatively large angle of deviation, good binocular potential, and no underlying neurological disease [5]. Type 1 AACE (Swan type) follows occlusion or loss of vision in one eye secondary to injury or disease [23]. Type 2 AACE (Franceschetti type) is characterized by acute onset of a relatively large angle of comitant esotropia and diplopia [3].…”

Section: Discussionmentioning

confidence: 99%

Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?

Turan

Kansu

2016

Journal of Ophthalmology

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Objectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings of patients presented with AACE of undetermined etiology with a minimum follow-up of 1 year were retrieved from the medical records and the results analyzed. Results. A series of 9 esotropia cases (age range: 20–43 years) was reviewed. All patients had full duction and versions, without an A-pattern or V-pattern. All patients had esotropia for distance and near. Neurological evaluation in all cases was normal. Among patients, 3 were treated with prisms, 4 were treated with strabismus surgery, and 1 was treated with botulinum toxin injections; 1 patient declined treatment. In treated patients posttreatment sensory testing indicated restoration of binocularity that remained stable throughout follow-up of 1–9 years. The patient that declined treatment had binocular function with base-out prisms. Conclusion. Acute onset esotropia may be seen without a neurological pathology in adults. Good motor and sensory outcomes can be achieved in these patients with AACE of undetermined etiology via surgical and nonsurgical methods.

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Section: Discussionmentioning

confidence: 99%

Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?

Turan

Kansu

2016

Journal of Ophthalmology

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“…Earlier investigators have reported acute acquired comitant esotropia occurring either immediately or soon after occlusion for anisometropic amblyopia . Lee and Kim reported an eight‐year‐old patient with anisometropic amblyopia and orthophoria who developed esotropia five months after terminating full‐time opaque occlusion.…”

Section: Discussionmentioning

confidence: 99%

“…When acute acquired comitant esotropia develops following occlusion therapy for anisometropic amblyopia, it is more likely to occur with anisohyperopic amblyopia, the esotropic eye likely having the larger ametropia . We report acute acquired comitant esotropia occurring in a child treated for severe anisomyopic amblyopia.…”

mentioning

confidence: 90%

“…Although frequently idiopathic, acute acquired comitant esotropia can be caused by interruption of binocular vision due to prolonged monocular eye lid closure or occlusive treatment for anisometropic amblyopia, decompensation of pre‐existing esophoria or small‐angle esotropia, loss of vision in one eye secondary to injury or disease, mild to moderate uncorrected myopia in adults, accommodative spasm and intracranial pathology . The last may present with additional signs and symptoms, such as abducting nystagmus, papilloedema, headaches, unsteadiness, as well as inability to achieve fusion with corrective prisms or the synoptophore .…”

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confidence: 99%

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Atypical presentation of acute acquired comitant esotropia

Rutstein

Lee

Zimmerman

2017

Clinical and Experimental Optometry

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“…Refractive accommodative esotropia typically presents during early childhood, having its usual onset between 18-48 months of age [1][2]. The onset of refractive accommodative esotropia in the second or third decade is uncommon, if it is not precipitated by a brief period of occlusion [3][4][5][6].Children usually present for treatment of a sudden onset of esotropia. Complaints of symptoms of refractive accommodative esotropia in young children are uncommon and for this reason ophthalmologists are not familiar to these complaints.…”

Section: Introductionmentioning

confidence: 99%

Atypical Onset of Refractive Accommodative Esotropia: A Case Report

Koç¹

2014

CMR

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A 27 year old female presented with symptoms of diplopia, headache, ptosis, ataxia, and macropsia. She was subjected to extensive and repetitive medical evaluations on 3 separate occasions over a 2 year period resulting in a delay in diagnosis, expense of the evaluations and unnecessary treatment. Ophthalmologic consultation uncovered a delayed onset of refractive accommodative esotropia. Signs and symptoms of refractive accommodative esotropia can simulate other neurologic diseases in adults.

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Esotropia Following Occlusion

Cited by 46 publications

References 6 publications

Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?

Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?

Atypical presentation of acute acquired comitant esotropia

Atypical Onset of Refractive Accommodative Esotropia: A Case Report

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