1991
DOI: 10.1016/0022-3468(91)90704-w
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Esophageal atresia, choanal atresia, and dysautonomia

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Cited by 14 publications
(10 citation statements)
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“…One or more of these dysautonomic features are found in nearly all patients with esophageal atresia [1][2][3], suggesting that autonomic disturbances and esophageal atresia may be manifestations of maldevelopment of cephalic neural crest derivates (neurocristopathy) [3]. This theory is supported by the findings that about three fourths of patients with esophageal atresia, such as the present patients with asymmetric sweating and/or flushing, present a neural crest related cardiovascular anomaly [11] and an asymmetric facial defect [3].…”
Section: Discussionmentioning
confidence: 72%
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“…One or more of these dysautonomic features are found in nearly all patients with esophageal atresia [1][2][3], suggesting that autonomic disturbances and esophageal atresia may be manifestations of maldevelopment of cephalic neural crest derivates (neurocristopathy) [3]. This theory is supported by the findings that about three fourths of patients with esophageal atresia, such as the present patients with asymmetric sweating and/or flushing, present a neural crest related cardiovascular anomaly [11] and an asymmetric facial defect [3].…”
Section: Discussionmentioning
confidence: 72%
“…The diagnosis of hyperhidrosis was made when the infant's bed sheet or clothes were wet from dripping sweat [1][2][3]. Hyperthermia was diagnosed in those infants who presented recurrent episodes of high body temperature without clinical or laboratory evidence of infection [1][2][3]. Apparent life-threatening episode (ALTE) was defined as respiratory arrest which prompted resuscitation maneuvers [1][2][3].…”
Section: Methodsmentioning
confidence: 99%
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“…During the ventilatory period characterised by severe glossoptotic pharyngeal obstruction we found: (1) a further decrease in the breathing rate; (2) increasing obstructed inspiratory efforts (equivalent of Muller's manoeuvre) often associated with stridor; (3) increasing obstructed expiratory efforts (equivalent of Valsalva's manoeuvre); and (4) retarded expiratory flow often associated with audible grunting. These findings are similar to those observed in children and adults with sleepapnoea syndrome.…”
Section: Discussionmentioning
confidence: 88%
“…10 In these infants, different types of mechanical airway obstruction, by increasing the negative intrathoracic pressure due to increased inspiratory efforts, offer evidence of an underlying dysautonomic control of upper airway dilating muscles, responsible for a collapse of soft pharyngeal airways. [10][11][12] Support for this concept comes from the good results obtained in a few infants with repaired EA and AO/ALTE syndrome treated by glossopexy, which stabilizes the upper airway by avoiding, to a certain extent, the falling back of the tongue. 13,14 At present, in infants with EA, there is still controversy as to whether overactive laryngeal chemoreflex, intrathoracic TC, and dysautonomic control of pharyngeal airway patency are independent or interrelated pathogenic factors.…”
Section: Introductionmentioning
confidence: 99%