Erratum to: Resolution of ST deviation after myocardial infarction in patients with and without sleep-disordered breathing

Sterz, Ulrich; Büchner, Stefan; Hetzenecker, Andrea; Satzl, Anna; Debl, Kurt; Luchner, Andreas; Hüsser, Oliver; Hamer, Okka W.; Fellner, Claudia; Zeman, Florian; Maier, Lars S.; Arzt, Michael

doi:10.1007/s11818-018-0165-5

Cited by 6 publications

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“…Patients with acute MI underwent CMR and polysomnography 3–5 days and 12 weeks after PCI [ 15 , 17 , 18 ]. In a subset of STEMI patients with analyzable [ 19 ] and available routine electrocardiograms (ECG) before PCI and > 24 h after PCI ( n = 27), a non-prespecified sub-analysis of ST-deviations was performed as described previously [ 20 ].…”

Section: Methodsmentioning

confidence: 99%

“…MI was assumed, if the signal intensity of hyperenhanced myocardium on delayed enhancement imaging was > 5 standard deviations above the mean signal intensity of the remote region and MVO was defined as a hypoenhanced region within the infarcted myocardium [ 24 , 25 ]. Infarct size and MVO were expressed as a percentage of the total left ventricular myocardial volume [ 20 ], when not stated otherwise. The MSI represents the difference between the area at risk and the final MI size highlighting the amount of myocardium which can be saved [ 15 ].…”

Section: Methodsmentioning

confidence: 99%

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The impact of epicardial adipose tissue in patients with acute myocardial infarction

et al. 2021

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Aims Epicardial adipose tissue (EAT) has been linked to impaired reperfusion success after percutaneous coronary intervention (PCI). Whether EAT predicts myocardial damage in the early phase after acute myocardial infarction (MI) is unclear. Therefore, we investigated whether EAT in patients with acute MI is associated with more microvascular obstruction (MVO), greater ST-deviation, larger infarct size and reduced myocardial salvage index (MSI). Methods and results This retrospective analysis of a prospective observational study including patients with acute MI (n = 54) undergoing PCI and 12 healthy matched controls. EAT, infarct size and MSI were analyzed with cardiac magnetic resonance imaging, conducted 3–5 days and 12 weeks after MI. Patients with acute MI showed higher EAT volume than healthy controls (46 [25.;75. percentile: 37;59] vs. 24 [15;29] ml, p < 0.001). The high EAT group (above median) showed significantly more MVO (2.22 [0.00;5.38] vs. 0.0 [0.00;2.18] %, p = 0.004), greater ST-deviation (0.38 [0.22;0.55] vs. 0.15 [0.03;0.20] mV×10−1, p = 0.008), larger infarct size at 12 weeks (23 [17;29] vs. 10 [4;16] %, p < 0.001) and lower MSI (40 [37;54] vs. 66 [49;88] %, p < 0.001) after PCI than the low EAT group. After accounting for demographic characteristics, body-mass index, heart volume, infarct location, TIMI-flow grade as well as apnea–hypopnea index, EAT was associated with infarct size at 12 weeks (B = 0.38 [0.11;0.64], p = 0.006), but not with MSI. Conclusions Patients with acute MI showed higher volume of EAT than healthy individuals. High EAT was linked to more MVO and greater ST-deviation. EAT was associated with infarct size, but not with MSI. Graphic abstract

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