Erratum: Canonical Wnt signalling regulates nuclear export of Setdb1 during skeletal muscle terminal differentiation

Abstract

Correction to: Cell Discovery (2016) 2, 16037; doi:10.1038/celldisc.2016.37; Published 18 October 2016 In the initial published version of this article, an error was made in Figure 6c, where the titles of x axis and y axis were left out. The corrected Figure 6c is now attached in this corrigendum. All figures and text description other than this remain the same as initially published.

Targets of histone H3 lysine 9 methyltransferases

“…H3K9me3 is also important for maintaining T H 2 lymphocytes since SUV39H1 participates in T H 1-specific gene repression and SETDB1 is required for maintaining stable lineage commitment ( Allan et al, 2012 ; Adoue et al, 2019 ). SETDB1-dependent H3K9me3 also represses adipogenic master regulatory genes until differentiation is required and regulates cell fate decisions in murine neurogenesis, myogenesis, and oligodendrocyte differentiation ( Tan et al, 2012 ; Liu et al, 2015 ; Matsumura et al, 2015 ; Beyer et al, 2016 ; Jiang et al, 2017 ). Loss of G9a during myogenesis and haematopoiesis results in derepression of lineage inappropriate genes and in the case of myogenesis, cell cycle regulators are also derepressed ( Chen et al, 2012 ; Rao et al, 2016 ).…”