Epilepsie

Scholz, W.; Hager, Helle Borgstrøm

doi:10.1007/978-3-662-38098-7_5

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Hippocampal Sclerosis1

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2009

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“…His concept allowed him to preserve the term “genuine epilepsy,” taken over from Gowers, even for patients diagnosed with hypoxic ischemic brain lesions. Spielmeyer (1925), whose focus was neuropathologic research on ischemia, and later his student Scholz (Scholz, 1951; Hager, 1956), Scholz and Hager (1956) provided evidence that strengthened the case for seizures being the cause of ischemic hypoxic lesions (Scholz, 1951). Spielmeyer (1927, 1928) saw analogies between the distribution pattern of the lesions in epilepsy patients and the distribution patterns of global ischemia.…”

Section: Hippocampal Sclerosismentioning

confidence: 99%

Clinical neuropathology of the epilepsies in the 100 years of the ILAE (1909–2009)

Meencke

2009

Epilepsia

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SUMMARYThe paper describes the history of the clinical neuropathology of the epilepsies in the last hundred years from microscopy to molecular neuropathology. The main focus is on the concepts of hippocampal sclerosis and its discussion of causative lesion or consequence of seizures, and the concept of developmental disturbances in respect to general epileptogenicity. Clinical neuropathology remains an important discipline in the future of epileptology and brain research especially in the area of molecular genetics. Neuropathology will help to understand the stages of epileptogenesis and the factors responsible for the progressive nature of the disease.

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Section: Hippocampal Sclerosismentioning

confidence: 99%

Clinical neuropathology of the epilepsies in the 100 years of the ILAE (1909–2009)

Meencke

2009

Epilepsia

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show abstract

Epilepsie

Cited by 1 publication

References 166 publications

Clinical neuropathology of the epilepsies in the 100 years of the ILAE (1909–2009)

Clinical neuropathology of the epilepsies in the 100 years of the ILAE (1909–2009)

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