Enzyme Changes within Muscle Fibers in Genetic and Nutritional Muscular Dystrophy

Tureen, Louis L.; Simons, R. D. G. P.

doi:10.3181/00379727-129-33327

Cited by 4 publications

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“…When this source of energy also fails, myolysis starts. The same increased LDH activity in enlarged muscle fibres was also found by Tureen and Simons [11] in chickens with genetic muscular dystrophy. …”

Section: Resultssupporting

confidence: 66%

Long-Term Dose-Response Effects of Vitamin E in Rats

Jager¹

1972

Ann Nutr Metab

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In order to investigate whether and to what extent the spontaneous in vitro haemolysis test is an indicator of vitamin E nutriture, six groups of 8 male rats were kept for 22 months on diets containing 0.5, 1.5, 2.5, 4.5, 8.5 or 16.5 mg D-α-tocopheryl acetate/kg food. The severity of myopathy in the leg muscles was correlated to the vitamin E content of the diet and to the percentage of in vitro haemolysis. More than 70% haemolysis appeared to indicate a state of vitamin E deficiency, associated with severe pathological changes in the skeletal muscles. Weight gain decrease and testis degeneration occurred only at the lowest dose of vitamin E. No relation existed between mortality and the doses of vitamin E given. Complete myolysis of a muscle cell was found to be preceded by a stage of swelling, in which a strong activity of lactic dehydrogenase was observed.

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Section: Resultssupporting

confidence: 66%

Long-Term Dose-Response Effects of Vitamin E in Rats

Jager¹

1972

Ann Nutr Metab

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“…The prec7se mechanisms by which selenium, sulfur amino acid, and vitamin E deficiency cause muscle damage are not known (56). The morphology of the muscle and AChE pattern indicates it is not similar to inherited muscular dystrophy (57).…”

Section: Focal Myopathy Of the Turkeymentioning

confidence: 99%

Developmental and Maturational Aspects of Inherited Avian Myopathies

Wilson

1990

Experimental Biology and Medicine

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Three inherited abnormalities of muscle growth of poultry are an inherited muscular dystrophy of the chicken, deep pectoral myopathy of turkeys and broilers, and focal myopathy of turkeys. The major features of each are described and compared. Cellular and molecular bases of dystrophy of the chicken and treatments to alleviate the disorder are discussed. The pathologic progression and anatomical basis for deep pectoral myopathy are presented. Evidence is given that focal myopathy of the turkey is a growth-dependent disorder. The implications of the idea that such disorders are partly consequences of selection are discussed in the context of the future needs of a poultry industry emphasizing processing of poultry meat.

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Multiple forms of acetylcholinesterase in nutritional and inherited muscular dystrophy of the chicken

Wilson

Viola

1972

Journal of the Neurological Sciences

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Enzyme Changes within Muscle Fibers in Genetic and Nutritional Muscular Dystrophy

Cited by 4 publications

References 0 publications

Long-Term Dose-Response Effects of Vitamin E in Rats

Long-Term Dose-Response Effects of Vitamin E in Rats

Developmental and Maturational Aspects of Inherited Avian Myopathies

Multiple forms of acetylcholinesterase in nutritional and inherited muscular dystrophy of the chicken

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