Enhanced antibacterial properties of enteric glial cells attenuate intestinal inflammation through the GABABR-mediated autophagy pathway
Ziteng Deng,
Jing Lan,
Jiaqi Wang
et al.
Abstract:Enterotoxigenic Escherichia coli (ETEC) infection is a severe threat to global public health because of its high morbidity and mortality among children and infants. Enteric glial cells (EGCs) are involved in host–bacteria communication. However, the mechanisms through which EGCs interact with ETEC remain unclear. We attempted to assess whether γ-aminobutyric acid type B receptor (GABABR) activation participated in EGC autophagy during Escherichia coli K88 (ETECK88) infection. Alterations in autophagy and EGC a… Show more
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