2019
DOI: 10.4067/s0718-85602019000100054
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Enfermedades cardiovasculares y cáncer: ¿dos entidades mutuamente relacionadas?

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Cited by 3 publications
(2 citation statements)
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“…52 Secondary activation of fibroblasts and endothelial cells causes synthesis of adhesion proteins (VCAM-1, ICAM-1, p-selectins), growth factors (PDGF and FGF), activated plasminogen inhibitor (PAI-1) and fatty acid synthetase rate (FAS) which perpetuates cell recruitment, foaming cells and angiogenesis. 53 This inflammatory microenvironment is similar to what happens in cancer. Epidemiological studies consider that up to 25% of cancers are triggered by a chronic inflammatory response.…”
Section: Shared Pathophysiological Mechanismsmentioning
confidence: 74%
“…52 Secondary activation of fibroblasts and endothelial cells causes synthesis of adhesion proteins (VCAM-1, ICAM-1, p-selectins), growth factors (PDGF and FGF), activated plasminogen inhibitor (PAI-1) and fatty acid synthetase rate (FAS) which perpetuates cell recruitment, foaming cells and angiogenesis. 53 This inflammatory microenvironment is similar to what happens in cancer. Epidemiological studies consider that up to 25% of cancers are triggered by a chronic inflammatory response.…”
Section: Shared Pathophysiological Mechanismsmentioning
confidence: 74%
“…Cardiovascular diseases are among the two leading causes of death in the world [ 75 ]. The n-3 PUFAs were shown to have numerous heart health benefits, preventing atherosclerosis by reducing total cholesterol concentrations, triglycerides [ 76 ] and blood pressure in patients with hypertension [ 77 ].…”
Section: Beneficial Effects Of N-3 Pufasmentioning
confidence: 99%