Endoscopic treatment of cerebrospinal fluid leaks with the use of lower turbinate grafts: a retrospective review of 125 cases

Abstract

In our hands, the success rate of endoscopic fistula repair was high, even in defects larger than 2 cm. Success rates may be further improved with accurate diagnosis of elevated intracranial pressure, a contributing factor to failure of spontaneous fistula repair.

Endoscopic Management of Cerebrospinal Fluid Rhinorrhea: The Charing Cross Experience

“…13 However, some publications cite sphenoid and ethmoid defects as more common. 12,15,24 We had two cases of iatrogenic CSF leaks secondary to endoscopic sinus surgery referred to our unit, whereas most cases were secondary to extensive base of skull or neurosurgery in contrast to the literature. 2 As a result, a larger proportion of sphenoid defects were noted in iatrogenic injuries, principally after trans-sphenoidal pituitary surgery.…”

Meningitis bacteriana aguda por Streptococcus agalactiae en una mujer no embarazada asociada a una fístula de LCR: comunicación de un caso

“…La técnica quirúrgica consiste en la colocación de un injerto que cubre la zona dehiscente que comunica la fosa craneal anterior con la fosa nasal. Para esto se utiliza preferentemente tejido del propio paciente como hueso, cartílago, colgajos de mucoperiostio, grasa o fascia así como también pegamentos biosintéticos 19 .…”

Current insights in CSF leaks: a literature review of mechanisms, pathophysiology and treatment options

“…In the chronic phase following the trauma, patients may experience intermittent nasal watery discharge, recurrent meningitis or brain abscess, headaches, salty/sweet taste in the rhinopharynx, hyposmia. The risk of developing posttraumatic recurrent meningitis varies from 12.5% to 50%, with a rate of 29.4% of neurological complications 16 . Being able to differentiate CSF rhinorrhea from other nasal secretions remains the cornerstone of diagnosing CSF fistulas.…”

“…In case of recurrent meningitis, patients should undergo evaluation for the defects that expose the meninges to the upper airways, regardless of the presence or absence of CSF leak 15 . In the acute phase, right after the trauma, patients may have epistaxis, chemosis, periorbital ecchymosis, visual impairment, anosmia or cranial nerve deficits (most frequently, I-III and V-VII) and meningitis 6,[16][17][18] . In the chronic phase following the trauma, patients may experience intermittent nasal watery discharge, recurrent meningitis or brain abscess, headaches, salty/sweet taste in the rhinopharynx, hyposmia.…”