2020
DOI: 10.1080/08820139.2020.1764577
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Endometriosis: What is the Influence of Immune Cells?

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Cited by 23 publications
(18 citation statements)
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“…According to current research, it is likely that immunological changes play a role in the development of endometriosis. 39 The reason why almost one in two endometriosis patients believed that the immune system was involved in the development of their illness might be due to the fact that this knowledge is spreading among patients. According to a paper on the coverage of endometriosis in the press, endometriosis is frequently associated with autoimmune diseases and environmental pollutants in women’s magazines.…”
Section: Discussionmentioning
confidence: 99%
“…According to current research, it is likely that immunological changes play a role in the development of endometriosis. 39 The reason why almost one in two endometriosis patients believed that the immune system was involved in the development of their illness might be due to the fact that this knowledge is spreading among patients. According to a paper on the coverage of endometriosis in the press, endometriosis is frequently associated with autoimmune diseases and environmental pollutants in women’s magazines.…”
Section: Discussionmentioning
confidence: 99%
“…Consequently, EM-circulating anti-endometrial antibodies could mask endometrial antigens. As a result, endometrial antigens might not be effectively recognized, with lost endometrial fragments remaining, potentially leading to ectopic establishment [ 83 , 88 , 89 ].…”
Section: Endometriosis and Inflammationmentioning
confidence: 99%
“…Aberrant expression of several cytokines by inflammatory cells, such as IL-1, IL-4, IL-6, IL-8, IL-10, IL-33, TNFα and growth factors, e.g., transforming growth factor (TGF-β), insulin-like growth factor (IGF-1), hepatocyte growth factor (HGF), epidermal growth factor (EGF), platelet-derived growth factor (PDGF), and vascular endothelial growth factor (VEGF), have been reported in EM [ 89 , 98 ]. Indeed, cytokines such as IL-8 and TNF-α are known to promote endometrial cell proliferation, endometrial adhesion, and angiogenesis.…”
Section: Endometriosis and Inflammationmentioning
confidence: 99%
“…These cytokines launch and enhance the inflammatory response, targeting the recruitment of various proinflammatory cells and mediators. Tumor necrosis factor and its receptors, TNFR1 and TNFR2, represent an extrinsic apoptosis pathway involved in endometriosis genesis, being implicated in inflammatory and endometrial repair [ 6 , 7 ].…”
Section: Introductionmentioning
confidence: 99%
“…The endocrine premature dendritic cells reach maturity and are carried through the lymphatic vessels in response to foreign antigens or various antigens on top of T cells from inflammatory targets. In endometriozic tissues, this physiological process is being modified and the population of CD83+ dendritic cells is significantly decreased, leading to endometrial antigen misrecognition by the circulating antiendometrial stromal cells [ 7 ].…”
Section: Introductionmentioning
confidence: 99%