Emergent Treatment of a Primary Aortoenteric Fistula with N-butyl 2-cyanoacrylate and Endovascular Stent

Outcome after endovascular stent graft repair of aortoenteric fistula: A systematic review

“…No conclusions on the associations between the type and configuration of the stent graft devises and the outcome can be drawn, because of the great variety of the stent grafts used at different anatomical sites. In 8 patients (19.51%) the endovascular management of the AEF was accompanied by repair of the contributing part of the gastrointestinal tract (fistula occlusion) with endoscopic, endovascular, or open surgical procedures; in one case a fibrin sealant was injected in the fistula tract endoscopically, 34 in another case the fistula was catheterized under angiographic control and was injected with N-butyl 2-cyanocrylate, 37 and in the remaining six cases the communication between the aorta and the bowel lumen was interrupted through a laparotomy or thoracotomy. 23,24,29,31,32,39 Adjunctive procedures other than fistula occlusion included diverting iliostomy, 41 CT-guided drainage of aortic sac, 41 psoas abscess drainage, 16 coilembolization of the aortic bifurcation, 28 and balloon dilatation of the stent graft to treat leakage.…”

Aortoesophageal Fistula Secondary to Placement of an Esophageal Stent: Emergent Treatment with Cyanoacrylate and Endovascular Stent Graft

“…16 Only two reports have described NBCA embolization for the AEF and aortoenteric fistula. [17][18][19] We suggest that NBCA embolization is useful for the prompt control of bleeding in hemodynamically unstable patients. Although NBCA was displaced after initial injection, we achieved successful embolization after additional injections.…”

Primary aorto-enteric fistula – a unique complication of poorly differentiated large B-cell lymphoma

“…About 22% of the cases involve the esophagus and the rest of the cases involve other parts of gastrointestinal tract [10]. Cancer can directly erode the bowel and vasculature wall that is further compromised by an aneurysm or inflammation induced destruction of a vessel wall by metal-loproteinases [11]. Proximity of the esophagus to the aorta and anatomic fixation of the 3 rd and 4 th portion of the duodenum makes these GI tract sections more vulnerable to invasive processes-even in the absence of aortic aneurysms [12].…”