Elevated levels of aldosterone in the amniotic fluid in two cases of congenital mesoblastic nephroma

Kotani, Tomomi; Sumigama, S.; Hayakawa, Hiroyoshi; Mano, Yukio; Tsuda, Hideo; Sugiyama, Chie; Kawachi, Fumitaka; Hironaka, Mikiko; Kato, Tatsuo; Kaneko, Kenzo; Hayakawa, Mika; Kikkawa, F.

doi:10.1002/uog.7637

Cited by 4 publications

(11 citation statements)

References 8 publications

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“…Congenital mesoblastic nephroma (CMN) is a rare pediatric tumor [1][2][3][4]12,13 . It is reported mainly in infants less than six months of age and rarely diagnosed beyond the age of three years.…”

Section: Discussionmentioning

confidence: 99%

“…The most common presentation is asymptomatic abdominal mass [1][2][3][4] . Hypertension could be an associated finding due to an increase in renin secretion from renal tissue infiltration [1][2][3][10][11][12][13] . This tumor could be suspected in-utero on ultrasonographic examination.…”

Section: Discussionmentioning

confidence: 99%

“…Neonatal hypertension, hematuria and hypercalcemia are clinical features associated with this tumor [5][6][7][8][9] . It has three variants: the classic form, the cellular or atypical form and the mixed kind 1,[6][7][8][9][10][11][12][13] . The classic form has no capsule, minimal cellularity and mitosis, no necrosis and no hemorrhage and has very excellent prognosis.…”

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confidence: 99%

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Stage III Atypical Cellular Mesoblastic Nephroma

Alhindi¹,

Shihadeh²

2016

BMB

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

mentioning

confidence: 99%

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Stage III Atypical Cellular Mesoblastic Nephroma

Alhindi¹,

Shihadeh²

2016

BMB

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“…It was reported that fetuses or neonates with CMN had an activated renin‐angiotensin‐aldosterone system . Infants with CMN showed an increased level of serum renin and aldosterone in the amniotic fluid . Hypertension occurred in the present neonate, and it immediately resolved after tumor resection.…”

Section: Discussionmentioning

confidence: 75%

“…Although, as mentioned above, ‘mirror’ did not hold true in terms of ‘edema’, ‘mirror’ may hold true in terms of ‘hypertension’ in this case. It was reported that fetuses or neonates with CMN had an activated renin‐angiotensin‐aldosterone system . Infants with CMN showed an increased level of serum renin and aldosterone in the amniotic fluid .…”

Section: Discussionmentioning

confidence: 99%

Maternal manifestation of Ballantyne's syndrome occurring concomitantly with the development of fetal congenital mesoblastic nephroma

Takahashi

Matsubara

Kuwata

et al. 2014

J of Obstet and Gynaecol

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Various fetal or placental disorders cause Ballantyne's (mirror) syndrome. For the first time, we report a maternal manifestation of Ballantyne's syndrome occurring concomitantly with the development of fetal congenital mesoblastic nephroma (CMN). In a pregnant woman with a CMN fetus, lung edema, hypertension, hyperthyroidism, and high serum human chorionic gonadotrophin level occurred, all of which characterize maternal manifestation of Ballantyne's syndrome. The fetus and placenta were devoid of 'edema', lacking 'triple edema', and thus this condition was not diagnosed as Ballantyne's syndrome; however, we considered this condition as the maternal manifestation of Ballantyne's syndrome. We performed emergent cesarean section at 28 weeks. Delivery acutely ameliorated maternal symptoms. Tumor was resected and was confirmed as CMN. Maternal manifestations of Ballantyne's syndrome, such as lung edema and hypertension, can occur in a mother with fetal CMN even without fetal and/or placental edema. The clinical course of this patient may suggest an etiology of Ballantyne's syndrome.

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