“…Th2‐specific chemokines, CCL17 and CCL22, are highly expressed in lung specimens, contributing to eosinophilic inflammation through the induction of interleukin (IL)‐4, IL‐5 and IL‐13 , which may provide clues to the immunopathogenesis of EP. CCL16 has important roles in the accumulation of H4‐expressing eosinophils and CCR1‐, 2‐, 3‐, 4‐ and 5‐expressing lymphocytes in EP . We previously demonstrated that dendritic cells (DC) and alveolar macrophages (AM) are the sources of CCL16 and CCL17 in EP .…”