Electrophysiologic studies in the syndrome of short P-R interval, normal QRS complex

Caracta, Anthony R.; Damato, Anthony N.; Gallagher, John J.; Josephson, Mark E.; Varghese, P. Jacob; Lau, Sun H.; Westura, Edwin E.

doi:10.1016/0002-9149(73)91037-0

Cited by 104 publications

(26 citation statements)

References 16 publications

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“…As discussed in more detail earlier , possible mechanisms are (1) increase in basal adrenergic tone, (2) local liberation of metabolites in response to ischemia, (3) structural changes in the AV node/AV node vicinity giving rise to complete or partial bypass of the impulses (Abinader and Shahar, 1980;Cataracta et al, 1973) which normally pass through the AV node, and (4) changes in AV conduction brought forward by altered autonomic nervous activity elicited by myocardial ischemia (Bishop and Peterson, 1978; Mallani ef al., 1969; Sole et al, 1980).…”

Section: Pathophysiologic Considerationsmentioning

confidence: 99%

Natural course of a prolonged pr interval and the relation between pr and incidence of coronary heart disease. A 7‐year follow‐up study of 1832 apparently healthy men aged 40–59 years

Erikssen

Otterstad

1984

Clinical Cardiology

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Summary: During a baseline cardiovascular survey PR was measured in a strictly standardized way in 1832 men aged 40-59 years, free from coronary heart disease (CHD). Of 1758 men still alive, 1585 underwent an identical follow-up study 7 years later. A total of 1570 were in sinus rhythm. The following findings were made: (1) Baseline and follow-up prevalence of a prolonged PR ( 2 0 . 2 2 s) was identical (5.3 vs. 5.4%). (2) Only 60% of restudied men with a prolonged PR also had prolonged PR at follow-up. (3) Only 1 of 98 with a prolonged baseline PR had a more advanced AV block at follow-up, whereas an additional 4 had conditions which might influence the AV node (1 Bechterew's disease and 3 mild aortic valve stenosis). (4) The incidence of all CHD events found during the follow-up study (CHD deaths, myocardial infarction, angina pectons, and pathologic exercise ECGs) was moderately but significantly lower in men with a prolonged PR than among men with a PR 1 0 . 2 1 s. Thus a prolonged PR is rarely an indicator of impending, more severe conduction disturbances; it is mostly a benign, functional find- ing in middle-aged men free from overt heart disease and is not positively associated with CHD. Rather PR may be moderately and inversely associated with latent CHD.

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Section: Pathophysiologic Considerationsmentioning

confidence: 99%

Natural course of a prolonged pr interval and the relation between pr and incidence of coronary heart disease. A 7‐year follow‐up study of 1832 apparently healthy men aged 40–59 years

Erikssen

Otterstad

1984

Clinical Cardiology

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“…They interpreted their data to suggest a partial A-V nodal bypass via a posterior internodal tract. Similarly, Caracta et al [3], in their study of 18 patients with a short P-R interval (> 0.12 sec) and normal QRS com plex, noted 2 subjects who had short A-H intervals (70 msec) during sinus rhythm. No significant increase in A-H intervals occurred in these 2 sub jects with atrial pacing.…”

Section: Discussionmentioning

confidence: 76%

His Bundle Electrocardiography in the Wolff-Parkinson-Syndrome

Gomes¹,

Haft²

1977

Cardiology

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His bundle electrographic studies with atrial pacing in 2 of 8 patients with the Wolff-Parkinson-White (WPW) syndrome demonstrated evidence for the combination of a James tract and Mahaim fibers. During normal sinus rhythm these 2 patients had P-H intervals of 65 and 75 msec and H-Q intervals of 15 and 35 msec, respectively. On atrial pacing, the P-H intervals showed a small increase with the H-Q intervals remaining constant. The QRS duration, δ-wave duration and P-J intervals remained relatively constant. These observations strikingly contrasted with the findings typical of Kent-type conduction in the remaining 6 patients in whom during sinus rhythm, the His depolarization occurred simultaneously with the onset of the (δ -wave on the surface ECG. During atrial pacing (ranges 85–174/min), in these 6 patients, there was a progressive increase in the duration of the δ -wave, gradual increase in the P-H and Q-H intervals, and an increase in the QRS duration. 5 of these 6 patients showed a progressive increase in the P-J intervals. These observations suggest a practical method for determining in the individual patient whether anomalous conduction is occurring via a Kent-type pathway or a combination of a James tract and Mahaim fibers arranged in series.

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“…"Gaps" in the propagation of a response may occur at ~ny level in the A-V conduction system (Agha et al, 1973;Camm et al, 1978;Gallagher et al, 1973;Moe et al, 1965;Waleffe et al, 1977;Wu et al, 1974). Unrecognised gaps in propagation due to proximal block may lead to an overestimation of refractoriness.…”

Section: Pitfalls In the Estimation And Interpretation Of Refractorinessmentioning

confidence: 99%

Untitled

1980

Clinical Cardiology

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Electrophysiologic studies in the syndrome of short P-R interval, normal QRS complex

Cited by 104 publications

References 16 publications

Natural course of a prolonged pr interval and the relation between pr and incidence of coronary heart disease. A 7‐year follow‐up study of 1832 apparently healthy men aged 40–59 years

Natural course of a prolonged pr interval and the relation between pr and incidence of coronary heart disease. A 7‐year follow‐up study of 1832 apparently healthy men aged 40–59 years

His Bundle Electrocardiography in the Wolff-Parkinson-Syndrome

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