1993
DOI: 10.1016/0033-0620(93)90017-8
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Electrocardiographic changes associated with acute cerebrovascular disease: A clinical review

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Cited by 80 publications
(61 citation statements)
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“…This shows that CK-MB is not the only marker of cardiac myocytolysis. Abnormally excessive levels of plasma catecholamine secondary to rapidly increase in intracranial pressure (13) , skeletal muscle damage as a result of a multiple injections (14) , negative caloric balance (15) can be some of the reasons attributed to the elevation of CK-MB levels in these patients. A study carried out in the University of Babylon, Iraq 2010 to determine the prognostic Significance of Troponin T and Creatine Kinase-MB in sera of patients with Acute Ischaemic Stroke (16) .…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…This shows that CK-MB is not the only marker of cardiac myocytolysis. Abnormally excessive levels of plasma catecholamine secondary to rapidly increase in intracranial pressure (13) , skeletal muscle damage as a result of a multiple injections (14) , negative caloric balance (15) can be some of the reasons attributed to the elevation of CK-MB levels in these patients. A study carried out in the University of Babylon, Iraq 2010 to determine the prognostic Significance of Troponin T and Creatine Kinase-MB in sera of patients with Acute Ischaemic Stroke (16) .…”
Section: Discussionmentioning
confidence: 99%
“…Physician regularly encounters patients with ECG changes associated with CNS lesions. Repolarization disturbances and dysrhythmias in acute stroke can be due to release of catecholamines, direct neural outcomes mediated from the CNS via neurons ending on the heart (2) , or co existing ischaemic heart disease (3) . Cardiac troponin is the important marker for myocardial damage (4) .…”
Section: Introductionmentioning
confidence: 99%
“…1 Arrhythmias and electrocardiographic changes are not uncommon in patients with acute intracranial vascular events due to neurohormonal changes, although the patient did not exhibit the electrocardiographic changes commonly observed, such as QT prolongation, depressed ST segments, and flat or inverted T waves. 1 The patient did not have a history of bradycardia before this hospitalization, making isolated sinus node dysfunction a highly unlikely explanation for her presentation. The ob- served gradual slowing of the heart rate and a previous history of vasovagal syncope suggested a reflex rather than intrinsic sinus node disease.…”
mentioning
confidence: 87%
“…Of these a few will demonstrate myocardial dysfunction and or damage. 4 Approximately half of patients whose cardiac arrest is due to an intracranial haemorrhage may collapse without complaining of a headache.…”
Section: Introductionmentioning
confidence: 99%