El papel de la radioterapia en el tratamiento del cáncer de ovario: revisión de la literatura

G, Benjamín Bianchi; P, Gonzalo Silva; C, Claudia Carvajal; B, Alejandro Santini

doi:10.4067/s0717-75262011000500013

Cited by 1 publication

(1 citation statement)

References 43 publications

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“…Although some reports have shown an important proportion of patients with anemia at the moment of diagnosis (Herrera, Thomas & Carlos, 2006). The authors attributed the result to the fact that the tumors of the patients under study were ulcerated and bleeding, similar to those reported by Carvajal Santana et al (2016) and Bianchi et al (2011). It should also be mentioned that the patients analyzed in the present study did not present significant ulcerations.…”

Section: Discussionsupporting

confidence: 90%

Presence of Papillomavirus DNA sequences in the canine transmissible venereal tumor (CTVT)

Ayala-Díaz

Jiménez-Lima

Ramírez-Alcántara

et al. 2019

PeerJ

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BackgroundThe canine transmissible venereal tumor (CTVT) or Sticker’s sarcoma is a neoplastic disease affecting dogs. This disease is presented as a tumoral mass in the genital organs of both, male and female individuals. Up to date, there is no clear evidence indicating a viral agent as the causative mediator for CTVT development.PurposeThe present work aims to analyze 21 samples from canines with CTVT for molecular identification of Papillomavirus DNA sequences. In addition, microbiological analysis, cytologic and histopathologic evaluations were also performed.ResultsAll patients showed no biochemical and microbiological alterations. Molecular analysis demonstrated the viral DNA presence in the samples using different primer sets. The MY primers amplified a 450 bp band in seven out of 21 samples (33%). The PVF and Fap64 primer set, targeting the L1 sequence of Canine Papillomavirus (CPV), showed positivity in 16 out of 21 samples (76%).ConclusionThese results support the possible causative association between CPV and CTVT; nevertheless, additional studies are required to uphold such statement. This work presents evidence indicating that a viral agent might be involved in the pathogenesis of CTVT and set the bases for a better understanding of the CTVT pathobiology.

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Section: Discussionsupporting

confidence: 90%