Ejaculatory Pain

Aasvang, Eske Kvanner; Møhl, Bo; Kehlet, Henrik

doi:10.1097/01.anes.0000270736.28324.61

Cited by 59 publications

(13 citation statements)

References 30 publications

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“…Pain sometimes lasted for 1 min after ejaculation and sometimes persisted for up to 2 days. In a great majority of patients, maximum pain was experienced on the external inguinal annulus and rarely spread to the scrotum and glans (19). In addition, the rate of patients exposed to postoperative painful ejaculation was much higher than expected.…”

Section: Discussionmentioning

confidence: 91%

“…In addition, the rate of patients exposed to postoperative painful ejaculation was much higher than expected. However, moderate-to-severe pain (2.8%) causes sexual dysfunction in patients and requires medical attention (19).…”

Section: Discussionmentioning

confidence: 99%

“…Aasvang et al reported that pressure-pain detection and tolerance thresholds were significantly lower in the dysejaculatory patients and all patients mentioned their maximum pain on the external inguinal annulus. (19). Although magnetic resonance examination may show spermatic cord pathology, cord edema, and increased spermatic cord caliber (23), there was no radiographic difference between chronic inguinal pain and dysejaculatory patients.…”

Section: Diagnosismentioning

confidence: 99%

See 2 more Smart Citations

An overlooked complication of the inguinal hernia repair: dysejaculation

Ece

Yılmaz

2018

Turk J Surg

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An overlooked complication of the inguinal hernia repair: DysejaculationThe objective of this study was to investigate the rate of post-herniorrhaphy dysejaculation in the current literature. A comprehensive search of PubMed, Medline, Google Scholar, and Google databases was performed using the keywords "groin hernia and chronic pain," "inguinal hernia and chronic pain," "dysejaculation," and "ejaculatory pain." The eligible studies were evaluated in terms of ejaculatory pain and surgical technique used. Ten studies with 122 patients were eligible for the analysis. The rate of ejaculatory pain for a total of 5521 patients was found to be 2.2%. The incidence of postoperative ejaculatory pain was found to be 2.1% following laparoscopic techniques and 1.1 % following open repair. Open techniques were not related to the increased frequency of dysejaculation. Sufficient data could not be obtained from the studies for the ejaculatory pain, and thus, no statistical evaluation was performed. Dysejaculation is a common cause of postoperative morbidity after inguinal hernia repair. Attention to technical details of the primary operation may reduce the incidence of dysejaculation.

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Section: Discussionmentioning

confidence: 91%

Section: Discussionmentioning

confidence: 99%

Section: Diagnosismentioning

confidence: 99%

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An overlooked complication of the inguinal hernia repair: dysejaculation

Ece

Yılmaz

2018

Turk J Surg

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“…This procedure is unusual in children with rhabdomyosarcoma due to the concern that it does not ensure oncological radicality [3], but no case of PO has been reported in series of adults undergoing this procedure for other tumors [4,5,6]. PO, instead, was reported after other surgical procedures, and particularly inguinal surgery [1,2]. …”

Section: Discussionmentioning

confidence: 99%

“…It can be associated with prostatitis, chronic pelvic pain syndrome, benign prostatic hyperplasia, ejaculatory duct obstruction, use of antidepressant medications and surgical procedures possibly as result of a nerve lesion [1,2]. …”

Section: Introductionmentioning

confidence: 99%

Painful Orgasm in an Adolescent after Seminal-Sparing Cystoprostatectomy: A Puzzling Symptom

Angelini¹,

Castagnetti²,

Rigamonti³

2014

Urol Int

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An 18-year-old boy, followed up after seminal-sparing cystectomy for bladder rhabdomyosarcoma, presented complaining of recurrent episodes of left scrotal/inguinal pain arising after orgasms. Full work-up ruled out disease recurrence, but showed enlarged seminal vesicles. Ligation of the vas deferens was unsuccessful. The patient was started on α-blockers to reduce vas contractions with improvement of symptoms. The possible pathophysiology and treatments of this symptom are discussed.

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