Effects of Spinal Cord Ischemia on Evoked Potential Recovery and Postischemic Regional Spinal Cord Blood Flow

Diretrizes para o tratamento cirúrgico das doenças da aorta da Sociedade Brasileira de Cirurgia Cardiovascular

“…Similarly, many experimental and clinical studies [47, 154,159,[178][179][180][181][182][183][184] have been developed trying to define the role of the motor-evoked or somatosensorial potentials, in monitoring the activity of the segmental medulla and in the prevention of the paraplegia, although with very variable results and with recommendations still not consensual in respect to the efficacy as an isolated method in the prevention of medullar ischemia [179].…”

Diretrizes para a cirurgia das doenças da aorta

“…Similarmente, vários estudos experimentais e clíni-cos 48,158,163,[186][187][188][189][190][191][192][193] têm sido desenvolvidos tentando definir o papel dos potenciais evocados motores ou somato-sensoriais, na monitorização da atividade da medula segmentar e na prevenção da paraplegia, embora com resultados muito variáveis, e de recomendação ainda não consensual, quanto à eficácia como método isolado na prevenção da isquemia medular 179 .…”

Intraoperative control by somatosensory evoked potentials in the treatment of cervical myeloradiculopathy. Results in 210 cases

“…For example, as little as 0.8°C of hypothermia increases latency [28]. This can be caused by a deeper level of anaesthesia [4, 10,32,33,44] or by haemodynamic disturbances such as hypotension, tachycardia or bradycardia, and also by local vascular abnormalities such as arterial occlusions or spasms, or external compression of vessels by a haematoma, bone graft, screw or bone or disk fragment [15,19,30]. It could be argued that we are testing the function of the posterior cord (sensory pathway) and not the more important and interesting anterior motor pathway.…”

“…In patients with cervical myelopathy, both ischaemic and compressive factors [15,30] are usual in the phys-iopathology, and the slightest decrease of collateral blood flows will cause rapid hypoxia of the posterior cords, which are very vulnerable to malfunction by being pressed against posterior anatomical structures. Like Epstein et al [10], we had no false-negatives in 210 monitored cases.…”