Effects of Personal Oral Hygiene and Subgingival Scaling on Bleeding Interdental Gingiva

Caton, Jack G.; Bouwsma, Otis J.; Polson, Alan M.; Espeland, Mark A.

doi:10.1902/jop.1989.60.2.84

Cited by 28 publications

(20 citation statements)

References 31 publications

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“…The aggressive periodontal diseases are clinically heterogeneous and include forms of disease clinically indistinguishable from other forms of periodontitis, and several forms of disease with a uniquely localized clinical presentation with significant destruction at a very early age (Caton et al 1989;Albandar et al 1997). These conditions show remarkable familial aggregation (Schenkein and Van Dyke 1994;Novak and Novak 1996).…”

Section: Introductionmentioning

confidence: 99%

Localized aggressive periodontitis is linked to human chromosome 1q25

Hastürk

et al. 2003

Hum Genet

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Localized aggressive periodontitis (LAP; previously known as localized juvenile periodontitis) is one of the rapidly progressive periodontal diseases. Certain forms of familial LAP show a simple Mendelian pattern of transmission. However, no gene mutation has been identified to be responsible for the LAP phenotype. As an initial step to identify a gene mutation associated with LAP, we have performed genetic linkage analysis with four multigenerational families exhibiting the LAP phenotype. Affected individuals in the families were identified based on clinical and laboratory criteria in an attempt to define a homogeneous phenotype, since the clinical presentation of LAP may represent a manifestation of a heterogeneous group of diseases. The LAP phenotype is linked to a DNA marker, D1S492, with LOD score 3.48, theta=0.00. The haplotype analysis of the chromosome interval associated with D1S492 indicates that a LAP locus is located between D1S196 and D1S533 on chromosome 1, covering about 26 million DNA basepairs. We have also examined the DNA sequence of prostaglandin-endoperoxide synthase 2 (PTGS2 or cyclooxygenase 2, COX2) since prostaglandin 2 (PGE2), the product of COX2, is upregulated in LAP patients and COX2 is located between D1S196 and D1S533. No mutation in COX2 was identified in the patients.

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Section: Introductionmentioning

confidence: 99%

Localized aggressive periodontitis is linked to human chromosome 1q25

Hastürk

et al. 2003

Hum Genet

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“…Despite the perceptible advantages of well-stimulated interproximal tissues [6, 7], very few indexes have tried to specifically measure these factors. Our review of the literature found only few indexes created to evaluate papilla inflammation [8–10].…”

Section: Introductionmentioning

confidence: 99%

A Proposed New Index for Clinical Evaluation of Interproximal Soft Tissues: The Interdental Pressure Index

Checchi

Montevecchi

Marucci

et al. 2014

International Journal of Dentistry

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The interdental pressure index (IPI) is introduced to specifically evaluate clinical interproximal-tissue conditions and assess the effect of interproximal hygiene stimulation. This index scores clinical responses of periodontal tissues to the apical pressure of a horizontally placed periodontal probe. It is negative when gingival tissues are firm, bleeding-free, and slightly ischemic by the stimulation; otherwise it is positive. The clinical validation showed high intraoperator agreement (0.92; 95% CI: 0.82–0.96; P = 0.0001) and excellent interoperator agreement (0.76; 95% CI: 0.14–1.38; P = 0.02). High internal consistency with bleeding on probing (κ = 0.88) and gingival index (Cronbach's α = 0.81) was obtained. Histological validation obtained high sensitivity (100%) and specificity (80%) for IPI+ toward inflammatory active form. The same results were recorded for IPI− toward chronic inactive form. IPI results as a simple and noninvasive method with low error probability and good reflection of histological condition that can be applied for oral hygiene motivation. Patient compliance to oral hygiene instructions is essential in periodontal therapy and IPI index can be a practical and intuitive tool to check and reinforce this important aspect.

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“…Patients were evaluated clinically by measuring Plaque index (PI) 29 , Gingival index (GI) 30 , Pocket depth (PD) 31 and Clinical attachment level (CAL) 32 at baseline, then at one, two and three months after the initiation of non surgical periodontal therapy.…”

Section: Clinical Outcomesmentioning

confidence: 99%

“…Probing depth (pocket depth) was measured from the gingival margin to the base of the periodontal pocket to the nearest mm (Caton, 1989 Statistical analysis: Data were statistically described in terms of mean and standard deviation (SD), frequency and percentage when appropriate. Data were explored for normality using Kolmogorov-Smirnov and Shapiro-Wilk tests.…”

Section: Probing Depth (Pd)mentioning

confidence: 99%

Untitled

2015

IJPSR

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Background:Periodontal disease is one of the two main and most prevalent oral diseases all over the world. Treatment strategies are diverse. To date, scaling and root planing (SRP) is still the gold standard non-surgical therapy for periodontitis. Systemically administered antibiotics can be used as an adjunct to SRP to improve the treatment outcome of periodontitis. Purpose: To compare clinical outcome of systemically administered doxycycline versus combination of amoxicillin and metronidazole as an adjunct to SRP in the management of chronic periodontitis patients. Methods: This study was conducted on forty-two moderate to severe generalized chronic periodontitis patients who received non-surgical periodontal therapy. Following SRP, patients were randomly allocated to one of the following groups; group (I) received Doxycycline (loading dose 200 mg and maintenance dose 100 mg/day) for fifteen days, group (II) received a combination therapy of amoxicillin and metronidazole (750 mg/day) for eight days and group III (control group) which was treated by SRP without administration of systemic antibiotic therapy. The periodontal parameters; Plaque index (PI), Gingival index (GI), Probing depth (PD) and Clinical attachment level (CAL) were examined for the assessment of the clinical outcome. Results: This study revealed that combination of amoxicillin and metronidazole resulted in significant reduction in PD and significant gain in CAL. Conclusion: The combination of Amoxicillin and Metronidazole at a dose of 750 mg/day for eight days had a significant effect on enhancing the clinical outcome of chronic periodontitis patients.

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Effects of Personal Oral Hygiene and Subgingival Scaling on Bleeding Interdental Gingiva

Cited by 28 publications

References 31 publications

Localized aggressive periodontitis is linked to human chromosome 1q25

Localized aggressive periodontitis is linked to human chromosome 1q25

A Proposed New Index for Clinical Evaluation of Interproximal Soft Tissues: The Interdental Pressure Index

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