Effects of daily rinsing and ingestion of fluoride solutions upon dental caries and enamel fluoride

Aasenden, R.; DePaola, P.F.; Brudevold, F.

doi:10.1016/0003-9969(72)90233-6

Cited by 98 publications

(39 citation statements)

References 10 publications

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“…APF 1.23% has a pH of 3.2-3.5, in which a low concentration of hydrogen dissolves the enamel surface to form CaF 2 . The application of APF induces fluoride deposition in hydroxyapatite and formation of fluorapatite, which can affect the bond strength (12).…”

Section: Introductionmentioning

confidence: 99%

Effect of fluoride solutions on the shear bond strength of orthodontic brackets

et al. 2012

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The aim of this in vitro study was to evaluate the shear bond strength of brackets after pre-treatment with different fluoride solutions. This study used 48 freshly extracted sound bovine incisors that were randomly assigned to 4 experimental groups (n=12). CG: (control) without treatment; NF: 4 min application of neutral fluoride; APF: application of 1.23% acidulated phosphate fluoride (APF) for 4 min; and SFV: application of 5% sodium fluoride varnish for 6 h. For each group, after surface treatment, prophylaxis of enamel and bracket bonding with Transbond XT composite resin (3M) were performed following the manufacturer's specifications. The shear bond strength was performed with a universal testing machine 24 h after fixing the brackets. The tooth surfaces were analyzed to verify the adhesive remnant index (ARI). Data were analyzed statistically by ANOVA and Tukey's test (α=0.05). There was statistically significant difference among the groups (p<0.0001). CG and NF groups presented significantly higher bond strength than APF and SFV. There was no significant difference between CG and NF or between APF and SFV (p>0.05). The analysis of ARI scores revealed that most failures occurred at the enamel-resin interface. It may be concluded that the pre-treatment of enamel with 1.23% APF and 5% SFV prior to fixing orthodontic brackets reduces shear bond strength values.

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Section: Introductionmentioning

confidence: 99%

Effect of fluoride solutions on the shear bond strength of orthodontic brackets

et al. 2012

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“…The fact that a cariostatic effect of fluoride could be obtained without a concomitant increase of fluoride in sound enamel [Brudevold et al, 1967;Aasenden et al, 1972;Shern et al, 1977] whereas enamel fluoride concentration increased in enamel undergoing a carious challenge [Hallsworth et al, 1971;Richards et al, 1977] became very important. By combining the above data with the results from theoretical and laboratory experiments on enamel solubility [Larsen, 1975;Larsen et al, 1976] it was tempting to suggest 'a possible explanation of the predominant cariostatic effect of fluoride' in 1981 [Fejerskov et al, 1981].…”

Section: Fluoride and Dental Cariesmentioning

confidence: 99%

Changing Paradigms in Concepts on Dental Caries: Consequences for Oral Health Care

2004

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Kuhn proposed in his Structure of Scientific Revolutions (1962) that the theoretical framework of a science (paradigm) determines how each generation of researchers construes a causal sequence. Paradigm change is infrequent and revolutionary; thereafter previous knowledge and ideas become partially redundant. This paper discusses two paradigms central to cariology. The first concerns the most successful caries-preventive agent: fluoride. When it was thought that fluoride had to be present during tooth mineralisation to ‘improve’ the biological apatite and the ‘caries resistance’ of the teeth, systemic fluoride administration was necessary for maximum benefit. Caries reduction therefore had to be balanced against increasing dental fluorosis. The ‘caries resistance’ concept was shown to be erroneous 25 years ago, but the new paradigm is not yet fully adopted in public health dentistry, so we still await real breakthroughs in more effective use of fluorides for caries prevention. The second paradigm is that caries is a transmittable, infectious disease: even one caused by specific microorganisms. This paradigm would require caries prevention by vaccination, but there is evidence that caries is not a classical infectious disease. Rather it results from an ecological shift in the tooth-surface biofilm, leading to a mineral imbalance between plaque fluid and tooth and hence net loss of tooth mineral. Therefore, caries belongs to common ‘complex’ or ‘multifactorial’ diseases, such as cancer, cardiovascular diseases, diabetes, in which many genetic, environmental and behavioural risk factors interact. The paper emphasises how these paradigm changes raise new research questions which need to be addressed to make caries prevention and treatment more cost-effective.

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“…Water fluoridation reduces the prevalence of dental decay by mecha nisms attributed to changes in the physical and chemical properties of en amel brought about by incorporation of fluoride ions in apatite crystals during the period of tooth formation [Brudevold and McCann, 1966;Jenkins, 1967], Topical fluorides also reduce caries prevalence [Torell and Erickson, 1967;Horowitz and Heifetz, 1970], but the efficacy of these preparations does not seem to be dependent upon their ability to drive fluoride into the tooth structure [Englander et al, 1967;Aasenden et al, 1972]. Topical fluorides are most effective in reducing cavita tion when used daily, i.e.…”

Section: Abstract the Percentage Of Streptococcus Mutans Abstractmentioning

confidence: 99%

Effect of Topical Acidulated Phosphate Fluoride on Percentage of Streptococcus mutans and Streptococcus sanguis in Plaque

Loesche¹,

Syed²,

Murray³

et al. 1975

Caries Res

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The percentage of Streptococcus mutans and Streptococcus sanguis in pooled occlusal plaque and in pooled approximal plaque following 8–10 daily applications of either an acidulated phosphate fluoride (APF) gel or a placebo gel was determined. S. mutans averaged about 9% of the colony forming units (CFU) in the occlusal plaque and 1% of the CFU in the approximal plaque prior to treatment. S. sanguis accounted for about 5% of the approximal isolates and 8% of the occlusal isolates prior to treatment. The APF treatment caused a persistent 45–75% reduction in the percentage of S. mutans in the occlusal samples but did not have an effect on the percentage of S. mutans in the approximal plaque. Great individual variability in the percentages of S. mutans prevented the demonstration of significant differences between the APF and placebo group when parametric statistical tests were employed. When ranking procedures were used, i.e. Kruskal Wallis, Willcoxon tests, the APF was found to have a specific antimicrobial effect against S. mutans in the occlusal plaque, but not in the approximal plaque. This effect was still apparent 12 weeks after the last treatment. There was no effect of the APF on the percentages of S. sanguis in either sample, except at one week after treatment, where the approximal samples showed a significant increase. The plaque and gingivitis scores in the APF group were significantly less than in the placebo group.

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Effects of daily rinsing and ingestion of fluoride solutions upon dental caries and enamel fluoride

Cited by 98 publications

References 10 publications

Effect of fluoride solutions on the shear bond strength of orthodontic brackets

Effect of fluoride solutions on the shear bond strength of orthodontic brackets

Changing Paradigms in Concepts on Dental Caries: Consequences for Oral Health Care

Effect of Topical Acidulated Phosphate Fluoride on Percentage of Streptococcus mutans and Streptococcus sanguis in Plaque

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