of iomatotropin and enhanced growth rate on the occurrence of osteochondrosis in pigs. Can. J. Anim' One hundred ind twenty yorkshiie pigs, equal numbers of gilts and barrows, with an initial average weight of 69 kg were us.ec in the present experiment. The animili weri treated with 2 -mg d -r of recombinant porcine somatotropin-(rpST). either b{ dqY injecti6n or by siow-release implant, while the control animals were treated with a sham implant. The daily injection of rpST increased daily weight gain (P j O.OOOfi and decreased feed/gain ratio (P < 0.0001) and daily feed intake (P < 0.0001). Barrows grew faster fi, : O.OZ\ andate more feeO (f : 0.0001) than-gilts. On reaching -100 kg, all pigs were slaughtered' and 60 pigs itwo from each pen) were randomly selected for carcass evaluation. Carcass lean content was increased (P < 0 0001) and carcass fat content was decreased (P < 0.0001) in animals treated with rpST by daily injection. Animals treated with rpST by the slowrelease implant were not different from the control animals in terms of growth performance artd carcass characteristics. No overt lameness was observed in any of the pigs in the experiment. However, rpST treatment, regardless.of the method of administration, increased the incidence and severity ot-osteoctronoiosis and reduced glycbsaminoglycan concentration in the cartilage ofthe weightbearing area of humeral and femoial condyles. These changes were independent of enhanced growth rate and carcass lean content'The reiults indicated that somatotropin treatment predisposes pigs to osteochondrosis.