Effect of Cortisone Acetate on Carcass Composition and Wool Characteristics of Weaned Lambs

Spurlock, Gillian; Clegg, M. T.

doi:10.2527/jas1962.213494x

Cited by 16 publications

(4 citation statements)

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“…It is not certain whether the increased intake was associated with the previously depressed appetite of the ewes. It appears that an increase in appetite can also occur in the absence of any prior depression of food intake, as Spurlock & Clegg (1962) have reported increases in the food intake of weaner sheep given 25, 100 or 300 mg. cortisone acetate thrice weekly for 103 days.…”

Section: Effects Of Cortisol On Food Intakementioning

confidence: 88%

The Influence of Cortisol on Food Intake and Glucose Metabolism in Sheep

Bassett¹

1963

Journal of Endocrinology

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Some metabolic effects of cortisol were investigated in non-pregnant ewes fed either ad lib. or on a restricted ration. Cortisol (25 mg./day) administered for a period of 21 days stimulated the voluntary food intake of fat ewes offered food ad lib. Higher levels of cortisol (50 and 75 mg./day) administered for similar periods subsequently had little additional effect on voluntary food intake, but an even greater cortisol dose (150 mg./day) resulted in a marked decline in voluntary food intake. Ewes fed a constant restricted ration did not refuse food at any time during cortisol administration. Urinary nitrogen excretion was increased during cortisol administration but sequential increases in the cortisol dose failed to increase urinary nitrogen excretion proportionately above the level attained during administration of 25 mg. cortisol/day. The blood glucose level was elevated progressively as the level of cortisol administered was increased. Similarly, tolerance for a glucose load was impaired progressively.Only very small changes in blood ketones and plasma free fatty acid levels were observed during cortisol administration.The changes in blood glucose during cortisol administration cannot be accounted for by changes in gluconeogenesis, and it is suggested that they reflect a progressive impairment of glucose utilization relative to the blood glucose level, though not a reduction in total glucose utilization.

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Section: Effects Of Cortisol On Food Intakementioning

confidence: 88%

The Influence of Cortisol on Food Intake and Glucose Metabolism in Sheep

Bassett¹

1963

Journal of Endocrinology

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“…Thyroidectomy of the sheep fetus retards growth and ossification, development of the skeleton and secondary skin follicles and causes muscular weakness (Erenberg et al, 1974 ;Thorburn, 1974) (Jost, 1977). Thyroidectomy after birth also retards the growth of lambs (Falconer and Draper, 1968) (Spurlock and Clegg, 1962 ;Clark et al, 1963 ;Carroll et al, 1963 in bull calves). This increase in adiposity could be due to hyperglycemia and hyperinsulinemia caused by the injections (Bassett and Wallace, 1967 ;Bassett, 1968).…”

Section: Introductionmentioning

confidence: 99%

Growth as affected by general hormonal factors and hormonal balances, and the limitations of such studies

Dauzier¹

1980

Reprod. Nutr. Dévelop.

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In neonatal life the correlations between plasma hormone concentrations and growth are more or less strict. These levels depend on the kinetic factors of hormone metabolism whose correlation to growth has been studied. The mode ot action of some hormonal treatments and the effect of selection on some hormonal balances are also discussed.In the third part of this report, a critical study indicates the limitations in investigations on growth endocrinology. Special attention has been given to the problem of hormone level fluctuations in relation to plane of nutrition, stress, light/dark cycle, as well as to the paucity of data on growth peptides and the limitations of assay methods.Introduction.

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“…Chez le mouton encore, des injections intraveineuses de glycine, d'alanine ou de lysine diminuent la prise alimentaire de concentrés de 12 à 30 p. 100 (Baile et Martin, 1971 (Bergen, 1972) L'octapeptide terminal de la cholécystokinine (CCK-OP) n'est vraisemblablement pas l'hormone physiologique de la satiété. Très récemment, Knoll (1979) Clegg, 1962), mais à condition que les moutons ne soient pas de gros mangeurs (Bassett, 1963 …”

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