Effect of Cardiac Dysrhythmia on Cerebral Perfusion

Sand, Bruce J.; Rose, Harold B.; Barker, Wiley F.

doi:10.1001/archsurg.1976.01360250063013

Cited by 11 publications

(2 citation statements)

References 12 publications

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“…Among these, PAF may reduce cardiac output and hence perfusion pressure to levels that compromise the cerebral circulation with consequent signs of hemodynarnic ischemia [5,9,15]. This phenomenon, known as autoregulation, prevents the onset of cerebral ischemia even when there are wide variations in perfusion pressure.…”

Section: Discussionmentioning

confidence: 99%

Transcranial Doppler evaluation of cerebral blood flow in patients with paroxysmal atrial fibrillation

et al. 1993

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Cerebral blood flow was measured by transcranial Doppler sonography (TCD) in 15 patients during and after an attack of paroxysmal atrial fibrillation. During the attack the mean flow velocity (MFV) was reduced significantly in the middle cerebral artery (p < 0.05) and nonsignificantly in the other arteries. The pulsatility index (PI) was reduced in all the arteries but not significantly. During the fibrillation phase the MFV and PI tended to decrease as the heart rate increased. None of the patients showed clinical signs of cerebral ischemia during the attacks. The absence of such signs despite the reduced cerebral blood flow in atrial fibrillation is probably due to the activation of mechanisms of autoregulation.

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Section: Discussionmentioning

confidence: 99%

Transcranial Doppler evaluation of cerebral blood flow in patients with paroxysmal atrial fibrillation

et al. 1993

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“…The ADQ may indicate cerebral ischaemia even if no significant change in arterial pressure is present during these events. Cardiac rhythms that may cause neurological harm may be rapidly detected, therefore, and possible neurological damage obviated [19].…”

Section: Effect Of Arterial Pressurementioning

confidence: 99%

Clinical Evaluation of the Augmented Delta Quotient Monitor for Intraoperative Electroencephalographic Monitoring of Children During Surgery and Cardiopulmonary Bypass for Repair of Congenital Cardiac Defects

Burrows

Volgyesi

James

1989

British Journal of Anaesthesia

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To assess the augmented delta quotient (ADQ) monitor as a monitor of cerebral function during cardiac surgery, we monitored during operation the electroencephalograms of 48 young subjects (aged 2 weeks to 19 yr). We found ADQ patterns produced by cardiopulmonary bypass, hypothermia and general anaesthetic agents correlated with those obtained from a compressed spectral array (CSA) monitor and could be differentiated from changes caused by cerebral ischaemia, except in the youngest group of patients (less than or equal to 18 months) undergoing deep hypothermia (19.4 (SD 0.8) degrees C nasopharyngeal). In all other age groups the ADQ proved to be a simple monitor of the adequacy of cerebral perfusion. Neurological deficit occurred only if the ADQ was abnormal during hypotension for a period exceeding 7 min. ADQ evaluation of cerebral function was limited by events which produced artificially normal ADQ readings such as low amplitude EEG activity and the described isoflurane effect that was demonstrated to occur in some cardiac patients. The results obtained by the ADQ were comparable to those obtained by compressed spectral array and the ADQ was easier to use and interpret.

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Cerebral microcirculatory changes during and following transient ventricular tachycardia in cats

Kobari

Fukuuchi

Tomita

et al. 1992

Journal of the Neurological Sciences

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Effect of Cardiac Dysrhythmia on Cerebral Perfusion

Cited by 11 publications

References 12 publications

Transcranial Doppler evaluation of cerebral blood flow in patients with paroxysmal atrial fibrillation

Transcranial Doppler evaluation of cerebral blood flow in patients with paroxysmal atrial fibrillation

Clinical Evaluation of the Augmented Delta Quotient Monitor for Intraoperative Electroencephalographic Monitoring of Children During Surgery and Cardiopulmonary Bypass for Repair of Congenital Cardiac Defects

Cerebral microcirculatory changes during and following transient ventricular tachycardia in cats

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