Dreaming: A Neurological View

Bischof, Matthias; Valko, Philipp O.

doi:10.1007/88-470-0550-7_15

Cited by 8 publications

(3 citation statements)

References 137 publications

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“…121 Most commonly RLS was bilateral, appeared within 1 week after stroke, and was accompanied by periodic limb movements (PLMs) in sleep. 149 Patients with strokes in the pontomesencephalic or mesencephalic tegmentum and in the paramedian 2 days after CVI 8 days after CVI 70 days after CVI thalamus can experience peduncular hallucinosis of Lhermitte, characterized by complex, often colorful, dreamlike visual hallucinations, particularly in the evening and at sleep onset ( Fig. 88-8).…”

Section: Insomniamentioning

confidence: 99%

“…154,155 Cessation or reduction of dreaming occurs in the Charcot-Wilbrand syndrome and is occasionally limited to alteration of the visual component of the dream (as in the original patient described by Charcot). 149,[157][158][159][160][161] Patients often, but not invariably, show deficient revisualization (referred to as visual irreminiscence), topographical amnesia, and prosopagnosia. 149,[157][158][159][160][161] Patients often, but not invariably, show deficient revisualization (referred to as visual irreminiscence), topographical amnesia, and prosopagnosia.…”

Section: Insomniamentioning

confidence: 99%

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Sleep and Stroke

Bassetti

2011

Principles and Practice of Sleep Medicine

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Section: Insomniamentioning

confidence: 99%

Section: Insomniamentioning

confidence: 99%

Sleep and Stroke

Bassetti

2011

Principles and Practice of Sleep Medicine

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“…Polysomnography parameters and sleep quality have been shown to be significantly worse in WD, both with and without RBD, as compared to healthy controls [ 15 ]. Diseases, which structurally affect the brain like stroke, traumatic brain injury, neoplasm, Parkinson's disease, Alzheimer's disease, fatal familial insomnia, and sporadic Creutzfeldt-Jakob disease, have been shown to have impact on dreaming [ 16 , 17 ]. In a study in WD, a single question on “presence of vivid dreams” was more often answered positively in patients than in controls, but the difference was not statistically significant [ 18 ].…”

Section: Introductionmentioning

confidence: 99%

Dream Recall Frequencies and Dream Content in Wilson’s Disease with and without REM Sleep Behaviour Disorder: A Neurooneirologic Study

Tribl

Trindade

Schredl

et al. 2016

Behavioural Neurology

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Objective. Violent dream content and its acting out during rapid eye movement sleep are considered distinctive for rapid eye movement sleep behaviour disorder (RBD). This study reports first quantitative data on dreaming in a cohort of patients with treated Wilson's disease (WD) and in patients with WD with RBD. Methods. Retrospective questionnaires on different dimensions of dreaming and a prospective two-week home dream diary with self-rating of emotions and blinded, categorical rating of content by an external judge. Results. WD patients showed a significantly lower dream word count and very few other differences in dream characteristics compared to age- and sex-matched healthy controls. Compared to WD patients without RBD, patients with WD and RBD reported significantly higher nightmare frequencies and more dreams with violent or aggressive content retrospectively; their prospectively collected dream reports contained significantly more negative emotions and aggression. Conclusions. The reduction in dream length might reflect specific cognitive deficits in WD. The lack of differences regarding dream content might be explained by the established successful WD treatment. RBD in WD had a strong impact on dreaming. In accordance with the current definition of RBD, violent, aggressive dream content seems to be a characteristic of RBD also in WD.

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Sleep and Stroke

Bassetti

2005

Semin Neurol

133

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More than 50% of stroke patients have sleep-disordered breathing (SDB), mostly in the form of obstructive sleep apnea (OSA). SDB represents both a risk factor and a consequence of stroke. The presence of SDB has been linked with poorer long-term outcome and increased long-term stroke mortality. Continuous positive airway presure is the treatment of choice for OSA. Oxygen and other forms of ventilation may be helpful in other (e.g., central) forms of SDB. SDB can improve spontaneously after stroke. About 20 to 40% of stroke patients have sleep-wake disorders (SWD), mostly in form of insomnia, excessive daytime sleepiness/fatigue, or hypersomnia (increased sleep needs). Depression, anxiety, SDB, stroke complications, and medications may contribute to SWD and should be addressed first therapeutically. Brain damage per se, often at thalamic or brainstem level, can be also a cause of persisting SWD. In these patients, hypnotics, dopeminergic agents, and stimulants (e.g., modafinil) can be attempted.

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Dreaming: A Neurological View

Cited by 8 publications

References 137 publications

Sleep and Stroke

Sleep and Stroke

Dream Recall Frequencies and Dream Content in Wilson’s Disease with and without REM Sleep Behaviour Disorder: A Neurooneirologic Study

Sleep and Stroke

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