Do angiotensin-induced changes in sheep renal venous blood and plasma composition indicate a reversal of glomerular filtration?

Intrarenal administration of angiotensin II does not moderate afferent renal nerve mediated cardiovascular reflexes in the anaesthetized rabbit

“…Large reductions in renal perfusion pressure induced by suprarenal aortic constriction , Thames & DiBona 1979 or renal artery constriction (Imanishi et al 1992) and more moderate reductions within the autoregulatory range (Osborn et al 1982, Imanishi et al 1992 have both been shown to result in renin release and an increase in angiotensin II in renal venous plasma. It has been suggested that this release of renin and subsequent generation of angiotensin II causes vasoconstriction of the efferent arteriole, increasing the filtration fraction and thus helping to maintain glomerular filtration rate, to compensate for the reduction in renal blood flow (Osborn et al 1987).…”