“…This topic was recently reviewed (Peak and Peak, 1989a), and the problems in identifying the reactive chemical species involved were discussed therein. Large oxygen-enhancement ratios for DNA damage and cytotoxicity of UV-A strongly implicate reactive oxygen species (such as the hydroxyl radical, singlet oxygen, superoxide anion, or H202 in UV-A effects (Peak et al, 1983(Peak et al, ,1985(Peak et al, , 1987Tyrrell and Pidoux, 1989). It has been speculated that H202 might be a cytotoxic agent produced within cells as a result of the absorption of UV-A (Eisenstark, 1987;Kramer and Ames, 1987 martano and Tuveson, 1985;Tyrrell, 1985).…”