“…In this regard, electrolytic and chemical lesions in this region, as well the injection of GABA agonists, produce a decrease in locomotion while electrical and chemical stimulation increases locomotor activity (Blake and Gladfelter, 1986, Gladfelter and Brobeck, 1962, Levy and Sinnamon, 1990, Marciello and Sinnamon, 1990, Shekhar and DiMicco, 1987, Sinnamon, 1984, Sinnamon, 1990, Sinnamon and Sklow, 1990, Sinnamon and Stopford, 1987, Wardas, et al, 1988. In addition, unit recordings have revealed non-hypocretinergic neurons that increase their firing rate in conjunction with motor activity (Lee, et al, 2005). Although hypocretinergic neurons seem to be primarily involved in somatomotor activity (Kiyashchenko, et al, 2002, Lee, et al, 2005, Martins, et al, 2004, Mileykovskiy, et al, 2005, Torterolo, et al, 2001, our results suggest that additional, as-yetunidentified neurons in this region may also be involved in this function.…”