Disappearing Pulse Syndrome Due to Myxomatous Degeneration of the Popliteal Artery

Barnett, Allen; Dugdale, L. M.; Ferguson, Ian

doi:10.5694/j.1326-5377.1966.tb73636.x

Cited by 14 publications

(2 citation statements)

References 12 publications

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“…On forced flexion, the narrowed arterial segment became displaced backwards, so that the artery lost its smooth curved course, and became M-shaped. Chevrier (1962) noted this effect in his case, and Barnett, Dugdale & Ferguson (1966), found that although the arteriogram looked normal on an A-P film, a stenosis was clearly seen in the lateral view. Operative findings, treatment and follow-up.…”

Section: Discussionmentioning

confidence: 88%

Arterial cystic degeneration

Savage

1972

Postgraduate Medical Journal

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Section: Discussionmentioning

confidence: 88%

Arterial cystic degeneration

Savage

1972

Postgraduate Medical Journal

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“…Particularly remarkable features in this report are the Echo-Doppler ultrasonographic findings clearly documenting the pathophysiological mechanism through which the popliteal artery adventitial cyst became symptomatic. Although previous investigators [ 4 - 8 ] described both the reduced distal pressure at the ankle, as measured on Doppler ultrasonography, and the angiographic features of popliteal stenosis after a symptom-provoking exercise test, they left the possible underlying mechanism unexplained. Our case report now suggests that an adventitial cyst of the popliteal artery can become symptomatic during muscle exertion when pressure within the fluid-filled cyst increases sufficiently to provoke hemodynamically significant endoluminal stenosis.…”

Section: Discussionmentioning

confidence: 99%

Doppler ultrasonography and exercise testing in diagnosing a popliteal artery adventitial cyst

Taurino

Rizzo

Stella

et al. 2009

Cardiovasc Ultrasound

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We describe popliteal arterial adventitial cystic disease which causes intermittent claudication in a young athletic man, with atypical manifestation, without loss of foot pulses on knee flexion nor murmur in the popliteal fossa. The findings obtained from Magnetic Resonance Imaging were nondiagnostic. The diagnosis resulted from Echo-Doppler ultrasonography along with peak exercise testing. Ultrasonography also provided useful physiopathological informations suggesting that a popliteal artery adventitial cyst can become symptomatic if muscle exertion increases fluid pressure within the cyst, enough to cause hemodynamically significant endoluminal stenosis. Rapid diagnosis is essential to prevent progressive claudication threatening limb viability. To guarantee this professional sportsman a reliable and durable outcome, instead of less aggressive management, we resected the involved arterial segment and interposed an autologous saphenous-vein graft.

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