2002
DOI: 10.1046/j.1445-2197.2002.02594.x
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Diplopia and headaches associated with cerebellopontine angle arachnoid cyst

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Cited by 11 publications
(26 citation statements)
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“…The mechanism of enlargement of the arachnoid cyst is explained by the osmotic pressure gradient between the inside and outside of the cyst wall, the secretion of fluid by the cyst wall, and possibly by a ball-valve mechanism or inadequate communication of the cystic contents with the subarachnoid space. Enlarging arachnoid cyst produces symptoms by direct compression of the surrounding brain and cranial nerves 3,7) . Patients with arachnoid cysts may present with ataxia, gait disturbance, headache, nausea, vomiting, character change, memory disturbance, symptoms mimicking transient ischemic attack, dysesthesias, dysphagia, nystagmus, and oscillopsia 1,12,19) .…”
Section: Discussionmentioning
confidence: 99%
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“…The mechanism of enlargement of the arachnoid cyst is explained by the osmotic pressure gradient between the inside and outside of the cyst wall, the secretion of fluid by the cyst wall, and possibly by a ball-valve mechanism or inadequate communication of the cystic contents with the subarachnoid space. Enlarging arachnoid cyst produces symptoms by direct compression of the surrounding brain and cranial nerves 3,7) . Patients with arachnoid cysts may present with ataxia, gait disturbance, headache, nausea, vomiting, character change, memory disturbance, symptoms mimicking transient ischemic attack, dysesthesias, dysphagia, nystagmus, and oscillopsia 1,12,19) .…”
Section: Discussionmentioning
confidence: 99%
“…Patients with arachnoid cysts may present with ataxia, gait disturbance, headache, nausea, vomiting, character change, memory disturbance, symptoms mimicking transient ischemic attack, dysesthesias, dysphagia, nystagmus, and oscillopsia 1,12,19) . These cysts also can cause dysfunction of specific CNs, including V (trigeminal neuralgia), VI (diplopia), VII (hemifacial spasm and facial nerve palsy), and VIII (hypacusia, tinnitus, and vertigo) 3,9,11,12,16) . However, to date there has been no report of a patient with glossopharyngeal neuralgia caused by an arachnoid cyst.…”
Section: Discussionmentioning
confidence: 99%
“…These cysts may become symptomatic because of (1) an increase in the osmotic gradient of the liquid contents; (2) creation of a valve mechanism between the arachnoid cyst and the subarachnoid space that leads to an increase in size and, as a consequence, symptoms, and (3) liquid secretion by the cyst wall, which alters its size [4]. Arachnoid cysts represent approximately 1% of all intracranial space-occupying lesions [1, 2, 3, 4, 5]. They are usually located in the middle cranial fossa, particularly in the temporo-sylvian area [1, 2, 3, 4, 5].…”
Section: Introductionmentioning
confidence: 99%
“…Arachnoid cysts represent approximately 1% of all intracranial space-occupying lesions [1, 2, 3, 4, 5]. They are usually located in the middle cranial fossa, particularly in the temporo-sylvian area [1, 2, 3, 4, 5]. The cerebellopontine angle (CPA) is the second most common location for arachnoid cysts, with a frequency of approximately 10% [2, 3, 4, 5, 6, 7].…”
Section: Introductionmentioning
confidence: 99%
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