Die Meleda-Krankheit

Hoede, Karl

doi:10.1007/bf01827916

Cited by 11 publications

(2 citation statements)

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“…A bluish-red demarcation zone between hyperkeratosis and normal skin has even been described in the dominant form (Costello & Gibbs 1967). Compared to Ma1 de Meleda the severe form differed only in the extension of the keratoderma and, in Sweden an incomplete form of this extremely rare inherited disorder might be possible (Hoede 1941). In previously performed studies of the Unna Thost variety, evidently recessive forms with a clinical picture differing from Ma1 de Meleda have been reported (Kansky et al 1982).…”

Section: Discussionmentioning

confidence: 97%

Two different clinical and genetic forms of hereditary palmoplantar keratoderma in the northernmost county of Sweden

Nielsen

1985

Clinical Genetics

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A follow‐up study of clinical and genetic observations, made on patients with hereditary palmoplantar keratoderma living in the northernmost county of Sweden (Norrbotten) in 1967, was performed. Two clinical types could be distinguished, a common form with an autosomal dominant mode of inheritance, corresponding to the description of the Unna Thost variety and a severe form with evidently an autosomal recessive inheritance. One of the patients with the severe form had a multilating palmoplantar keratoderma. Neither employment nor dermatophytosis influenced the severity of the hyperkeratosis in any of these two types. Occurrence of hereditary palmoplantar keratoderma together with other genodermatoses or dermatoses with a polygenic mode of inheritance was also found in this study.

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Section: Discussionmentioning

confidence: 97%

Two different clinical and genetic forms of hereditary palmoplantar keratoderma in the northernmost county of Sweden

Nielsen

1985

Clinical Genetics

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“…annähernd diffuse, scharfbegrenzte, 2. insei-und streifenförmige und 3. multipel-kleinherdförmige PPK . Die neueren Arbeiten von Kogoj (1934), Tappeiner (1937), Hoede (1941Hoede ( ) und llanhart (1947 haben ergeben, daß eine ganze Reihe von klinisch annähernd diffusen idiopathischen PPK sich im Gegensatz zur ebenfalls diffusen aber dom inant vererbten PPK vom Typus Unna-Thost rezessiv vererbt. Aus biologischen Gründen liegt es nahe, daß bei den erbbedingten K rankheiten der Erbgang als natürliches Einteilungsprinzip über dem klinischen Bild stellt.…”

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