Diagnostic significance of pulmonary hypertrophic osteoarthropathy

Vogl, A; Blumenfeld, Serge; Gutner, Leonard B.

doi:10.1016/0002-9343(55)90205-9

Cited by 62 publications

(13 citation statements)

References 20 publications

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“…However, some of the disorders described as systemic manifestations of carcinoma of the bronchus are otherwise notably rare. Moreover a partial or complete remission has been reported following treatment of the primary lung tumour in many of them: in hypertrophic pulmonary osteoarthropathy (Vogl, Blumenfeld, and Gutner, 1955); in the neuromuscular syndromes (Brain and Henson, 1958); in hypercalcaemia (Connor et al, 1956); in gynaecomastia (Hardy, 1960); and in the syndrome of hyponatraemia and renal sodium loss (Schwartz et al, 1957). This patient initially presented with severe joint pains.…”

Section: Discussionmentioning

confidence: 89%

Malabsorption syndrome associated with carcinoma of the bronchus

Wangel¹,

Deller²

1965

Gut

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Section: Discussionmentioning

confidence: 89%

Malabsorption syndrome associated with carcinoma of the bronchus

Wangel¹,

Deller²

1965

Gut

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“…On the other hand, patients can also present with features of adult Still's disease [36], sacroiliitis and spondylarthropathy [34], or RA [24]. Another manifestation is hypertrophic osteoarthropathy (Pierre-Marie-Bamberger), which can be seen in patients with various malignancies, including bronchogenic carcinoma [50]. Usually the lower extremities are involved [1] with characteristic clubbing of fingers and toes, which was not described in this case.…”

Section: Differential Diagnosismentioning

confidence: 94%

A 26-year-old woman with fever, migratory polyarthritis, and pericarditis subsequent to a T-cell lymphoma

Brouwer

Kluin-Nelemans

Niezen³

et al. 1997

Ann Hematol

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A 26-year-old woman was hospitalized for evaluation of persistent fever, migratory arthritis, and general malaise. Eighteen months before admission, Ki-1-positive T-cell lymphoma with inguinal lymphadenopathy, hepatosplenomegaly, and bone marrow localization (stage IV B) had been diagnosed. She received six cycles of CHVmP/BV consisting of cyclophosphamide, adriamycin, teniposide, prednisone, bleomycin, and vincristine. Six months later she developed skin lesions on her legs and chest, which later spread to her abdomen and extremities. A biopsy of one of the lesions showed perivascular infiltrates of mainly CD30 (Ki-1)-positive cells. A diagnosis of lymphomatoid papulosis (LP) was made. During the following weeks the lesions disappeared spontaneously. Shortly afterwards, she underwent an autologous bone marrow transplantation following BEAC (carmustine, etopiside, cytarabine, and cyclophosphamide) polychemotherapy.One year prior to admission a relapse of the skin lesions had occurred with fever, malaise, and lymphadenopathy. All of these symptoms disappeared without therapy. Follow-up chest X-rays, computed tomographic (CT) scans of the abdomen and thorax, and bone marrow biopsies showed no lymphoma. Five months before admission she had complained of pain and swelling of her left wrist, thumb, and ankle. Tendinitis was diagnosed. Fever then developed with increasing chest pain, dyspnea, and general malaise. A chest X-ray showed cardiomegaly with pleural fluid on the left side. An echocardiogram revealed pericardial effusion. In total, 650 ml of pericardial fluid were aspirated; all cultures including those for tuberculosis and fungi were negative. Cytological examination disclosed no lymphoma cells. Laboratory results revealed weakly positive antinuclear antibodies (ANA), without anti-double-stranded DNA (anti-dsDNA) antibodies. No antibodies against extractable nuclear antigen (ENA) or rheumatoid factor were detected. Serum complement levels were normal.

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“…These findings, however, were not constant in his series, and readings were normal in his three cases of hypertrophic pulmonary osteoarthropathy. The increase in blood flow, whatever its mechanism, has been established by later authors (Holling and Brodey, 1961;Vogl, Blumenfeld, and Gutner, 1955).…”

mentioning

confidence: 86%

Gross hypertrophic pulmonary osteoarthropathy in a 7-year-old child

Barclay¹,

Ogbeide²,

Grillo³

1970

Thorax

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Hypertrophic pulmonary osteoarthropathy in children is rare and usually associated with an underlying condition. A case of hypertrophic pulmonary osteoarthropathy secondary to a chronic post-measles lung abscess is presented. There was good radiological evidence of an advanced degree of periosteal reaction. This became much more obvious during an exacerbation of the illness. Films taken 10 months after resection of the affected lobe showed almost complete resolution of the bone condition. A feature that is well known in adults, but has not previously been reported in a child, was the exquisite tenderness of the affected bones. The literature is reviewed.

show abstract

Diagnostic significance of pulmonary hypertrophic osteoarthropathy

Cited by 62 publications

References 20 publications

Malabsorption syndrome associated with carcinoma of the bronchus

Malabsorption syndrome associated with carcinoma of the bronchus

A 26-year-old woman with fever, migratory polyarthritis, and pericarditis subsequent to a T-cell lymphoma

Gross hypertrophic pulmonary osteoarthropathy in a 7-year-old child

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