Diagnosis of &lt;b&gt;&lt;i&gt;Helicobacter pylori&lt;/i&gt;&lt;/b&gt; Infection by the Arrangement of Collecting Venules Using White Light Endoscopy: Evaluation of Interobserver Agreement

Garcés-Durán, Rodrigo; Galdin-Ferreyra, M; Delgado-Guillena, Pedro; Cuatrecasas, Míriam; Córdova, Henry; García-Rodríguez, Ana; Rodrigo-Calvo, María Teresa; Jimeno-Ramiro, Mireya; Araujo, Isis K.; Ginès, Pere; Llach, Josep; Fernández‐Esparrach, Glòria

doi:10.1159/000518100

Cited by 8 publications

(4 citation statements)

References 41 publications

(70 reference statements)

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“…The regular arrangement of collecting venules could be observed in the lower part of the stomach body and gastric angle under white-light endoscopy (▶ Fig. 1 a), consistent with an H. pylori-uninfected mucosal background [1,2]. On the anterior wall of the upper gastric body, a 15-mm slightly elevated (0-IIa) and whitish lesion (lesion A) was identified.…”

supporting

confidence: 57%

A unique case of well-differentiated gastric-type adenocarcinoma coexisting with a gastric adenocarcinoma of the fundic gland in a Helicobacter pylori-uninfected stomach

Dong,

Wu,

Xia

et al. 2023

Endoscopy

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supporting

confidence: 57%

A unique case of well-differentiated gastric-type adenocarcinoma coexisting with a gastric adenocarcinoma of the fundic gland in a Helicobacter pylori-uninfected stomach

Dong,

Wu,

Xia

et al. 2023

Endoscopy

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“…Besides the detection of H. pylori, pathological examination of the gastric mucosa also provides information on the degree and pattern of inflammation and the presence/absence of preneoplastic lesions. Additional biopsies are considered from the antrum and the body for culture-based AST of H. pylori in those patients with high epidemiologic risk factors for H. pylori infection and negative RAC pattern (57,58).…”

Section: Strong Recommendation (100% Of Agreement) High Quality Evidencementioning

confidence: 99%

Belgian consensus for Helicobacter pylori management 2023

Garcés-Durán¹,

Kindt²,

Kotilea³

et al. 2023

AGEB

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Helicobacter pylori (H. pylori) infection causes chronic gastritis, peptic ulcers and gastric cancer. Although H. pylori prevalence is decreasing worldwide, regional variations exist in Europe, with the lowest infection prevalence in Northern Europe, and the highest in Eastern and Southern Europe (1). Changes in the treatment recommendations and the increasing available evidence have justified the implementation of new recommendations since last Belgian consensus in 1998 (2). Several non-H. pylori Helicobacter species (NH.PYLORI-H), colonizing the stomach of domestic animals, also have the ability to cause gastric disease in humans, although to a lesser extent. These zoonotic NH. PYLORIH are not the subject of the current recommendations.

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“…Further research is required to confirm these findings. RAC provides high kappa values of intra-observer and inter-observer agreements of 0.88-0.91 and 0.74-0.79, respectively[ 21 , 68 ]; however, agreement between the other endoscopic findings needs to be clarified.…”

Section: Future Perspectives In Kyoto Cassificationmentioning

confidence: 99%

Kyoto classification of gastritis: Advances and future perspectives in endoscopic diagnosis of gastritis

Toyoshima¹,

Nishizawa²

2022

World J Gastroenterol

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This editorial provides an update of the recent evidence on the endoscopy-based Kyoto classification of gastritis, clarifying the shortcomings of the Kyoto classification, and providing prospects for future research, with particular focus on the histological subtypes of gastric cancer (GC) and Helicobacter pylori ( H. pylori ) infection status. The total Kyoto score is designed to express GC risk on a score ranging from 0 to 8, based on the following five endoscopic findings: Atrophy, intestinal metaplasia (IM), enlarged folds (EF), nodularity, and diffuse redness (DR). The total Kyoto score reflects H. pylori status as follows: 0, ≥ 2, and ≥ 4 indicate a normal stomach, H. pylori -infected gastritis, and gastritis at risk for GC, respectively. Regular arrangement of collecting venules (RAC) predicts non-infection; EF, nodularity, and DR predict current infection; map-like redness (MLR) predicts past infection; and atrophy and IM predict current or past infection. Atrophy, IM, and EF all increase the incidence of H. pylori -infected GC. MLR is a specific risk factor for H. pylori -eradicated GC, while RAC results in less GC. Diffuse-type GC can be induced by active inflammation, which presents as EF, nodularity, and atrophy on endoscopy, as well as neutrophil and mononuclear cell infiltration on histology. In contrast, intestinal-type GC develops via atrophy and IM, and is consistent between endoscopy and histology. However, this GC risk-scoring design needs to be improved.

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Diagnosis of <b><i>Helicobacter pylori</i></b> Infection by the Arrangement of Collecting Venules Using White Light Endoscopy: Evaluation of Interobserver Agreement

Cited by 8 publications

References 41 publications

A unique case of well-differentiated gastric-type adenocarcinoma coexisting with a gastric adenocarcinoma of the fundic gland in a Helicobacter pylori-uninfected stomach

A unique case of well-differentiated gastric-type adenocarcinoma coexisting with a gastric adenocarcinoma of the fundic gland in a Helicobacter pylori-uninfected stomach

Belgian consensus for Helicobacter pylori management 2023

Kyoto classification of gastritis: Advances and future perspectives in endoscopic diagnosis of gastritis

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