Diagnosis and Treatment of Neurocysticercosis

Coyle, Christina M.; Tanowitz, Herbert B.

doi:10.1155/2009/180742

Cited by 38 publications

(43 citation statements)

References 98 publications

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“…In vesicular neurocysticercosis (active) (Fig 8), a fully grown cyst (5-20 mm) or a cluster of cysts (racemose form) (23) is frequently found at the subarachnoid space near the gray matter-white matter junction or in the basal ganglia, cerebellum, brainstem, cisterns, or ventricular system. A 2-4-mm scolex can be seen in nearly 50% of cases (17).…”

Section: Classificationmentioning

confidence: 99%

“…The clinical presentation of patients with neurocysticercosis is highly variable and nonspecific and depends on the number, location, size, and stage of the parasites, as well as the degree of the host's inflammatory response (12,23). Furthermore, individual responses to the parasite differ.…”

Section: Clinical Presentationmentioning

confidence: 99%

“…In contrast, signs of meningeal irritation are uncommon (<2% of cases) (9). Symptoms may be delayed for several years or may remain subclinical, but some patients can present with life-threatening conditions (23).…”

Section: Clinical Presentationmentioning

confidence: 99%

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Neurocysticercosis: Radiologic-Pathologic Correlation

Kimura‐Hayama¹,

Higuera²,

et al. 2010

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Neurocysticercosis is a neurologic parasitic disease caused by the encysted larva of the tapeworm Taenia solium and is the most important parasitic disease of the human central nervous system. It is the most common cause of acquired epilepsy in endemic settings and constitutes a public health challenge for most of the developing world. Nowadays, however, as a result of globalization, neurocysticercosis is being seen more frequently in developed countries as well. Neurocysticercosis is acquired through fecal-oral contamination, and the disease course is complex, with two intermediate hosts (ie, pigs and humans) and a definitive host (humans). Traditionally, it has been classified into active and nonactive forms according to disease location. Radiologists must be aware of its imaging appearance, which is quite variable, as is the differential diagnosis. Imaging findings depend on several factors, including the stage of the life cycle of T solium at presentation; the number and location (ie, subarachnoid, cisternal, or intraventricular) of parasites; and associated complications such as vascular involvement (ie, arteritis with or without infarction), inflammatory response (ie, edema, gliosis, or arachnoiditis), and, in ventricular forms, degree of obstruction. Thus, the diagnostic approach, management, and prognosis for neurocysticercosis differ widely depending on the type of infection.

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Section: Classificationmentioning

confidence: 99%

Section: Clinical Presentationmentioning

confidence: 99%

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Neurocysticercosis: Radiologic-Pathologic Correlation

Kimura‐Hayama¹,

Higuera²,

et al. 2010

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“…Signs and symptoms are related to the number, size, stage, location of lesions and severity of the host's immune response to the parasites [10,11]. Seizures are the most common presentation in parenchymal neurocysticercosis (NCC) followed by headache [7].…”

Section: Clinical Presentationmentioning

confidence: 99%

A comprehensive review of imaging findings in human cysticercosis

et al. 2016

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“…Clinical expression of NCC might disclose a variable mixture of neurologic and psychiatric signs, even a chronic, diffuse brain dysfunction as dementia can be produced by NCC [6,15]. A single cysticercus located in the brainstem, or within the eye, or in the pituitary gland [16] may produce different and more severe clinical manifestations than various cysticerci scattered in brain parenchyma but not situated in eloquent neurologic sites [9].…”

Section: Clinical Manifestationsmentioning

confidence: 99%

Clinical Manifestations, Diagnosis, and Treatment of Neurocysticercosis

Sotelo

2011

Curr Neurol Neurosci Rep

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Neurocysticercosis (NCC) is the most frequent parasitic disease of the human brain. Modern imaging studies, CT and MRI, have defined the diagnosis and characterization of the disease. Through these studies the therapeutic approach for each case may be individualized with the aid of antihelmintics, steroids, symptomatic medicines, or surgery. The use of one or various therapeutic measures largely depends on the peculiar combination of number, location, and biological stage of lesions as well as the degree of inflammatory response to the parasites. Although there is not a typical clinical picture of NCC, epilepsy is the most frequent manifestation of parenchymal NCC, whereas hydrocephalus is the most frequent manifestation of meningeal NCC. Eradication of cysticercosis is an attainable goal by public education and sanitary improvement in endemic areas.

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Diagnosis and Treatment of Neurocysticercosis

Cited by 38 publications

References 98 publications

Neurocysticercosis: Radiologic-Pathologic Correlation

Neurocysticercosis: Radiologic-Pathologic Correlation

A comprehensive review of imaging findings in human cysticercosis

Clinical Manifestations, Diagnosis, and Treatment of Neurocysticercosis

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