Development of collagenomas during pregnancy

McClung, Amy A.; Blumberg, Mark; Huttenbach, Yve; Colome‐Grimmer, Maria; Raimer, S.

doi:10.1016/j.jaad.2005.01.003

Cited by 22 publications

(18 citation statements)

References 11 publications

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“…No successful therapeutic options have been reported, and connective tissue nevi and FCC lesions have persisted in all patients10. There is no malignant potential and most lesions do not equire treatment.…”

Section: Discussionmentioning

confidence: 99%

A Case of Familial Cutaneous Collagenoma

et al. 2011

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Familial cutaneous collagenoma is a rare hereditary disease that is inherited in an autosomal dominant pattern. It is characterized by early onset of multiple, skin-colored, sometimes hypopigmented cutaneous nodules, which initially show a symmetrical arrangement on the trunk, and later on the neck and upper limbs. We report on a case of a 45-year-old female who presented with multiple oval to round hypopigmented papules measuring 5~15 mm on her trunk. Histopathologically, the lesions showed an increased amount of collagen fibers and decreased, fragmented elastic fibers in the dermis. The skin lesions were diagnosed as familial cutaneous collagenoma and no treatment was administered. To the best of our knowledge, our case is the first reported case of familial cutaneous collagenoma (FCC) in the Korean literature.

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Section: Discussionmentioning

confidence: 99%

A Case of Familial Cutaneous Collagenoma

et al. 2011

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“…Uitto et al 10 showed that collagenoma almost exclusively consists of type I collagen and the underlying defect seemed to be a reduced production of collagenase in that location, and therefore a decreased local degradation of collagen. And some reports that the growth of collagenoma was influenced during pregnancy or puberty imply that hormone may be involved in the pathogenesis of this disorder 1,11 . No specific treatment is given in most cases 11 .…”

Section: Discussionmentioning

confidence: 99%

“…And some reports that the growth of collagenoma was influenced during pregnancy or puberty imply that hormone may be involved in the pathogenesis of this disorder 1,11 . No specific treatment is given in most cases 11 . Two cases of eruptive collagenomas were reported to be treated with intralesional steroids with transient flattening of the lesions 7,9 .…”

Section: Discussionmentioning

confidence: 99%

“…Our case has a different characteristic from the previously reported cases. Nine cases 4,9,[11][12][13][14][15][16][17] , in the English literature and two cases 18,19 in Korean literature have been reported to date. Most patients developed eruptive collagenoma mainly on the trunk, abdomen and upper extremities, whereas one patient developed lesions localized on the left back 16 .…”

Section: Discussionmentioning

confidence: 99%

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A Case of Eruptive Collagenoma on the Left Calf

et al. 2008

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Eruptive collagenoma is an acquired connective tissue nevus without family history. It is typically described as numerous small papules or nodules on the trunk and arms with histopathological features of decreased or degenerated elastic fibers. We report a case of a 16-year-old male who presented with multiple asymptomatic 2 to 5 mm sized yellowish grouped papules on the left calf. Histopathologically, the lesion showed thickened homogenized collagen fibers highlighted by Masson trichrome stain and decreased and fragmented elastic fibers stained by Verhoeff-van Gieson stain. The skin lesion was diagnosed as eruptive collagenoma and no treatment was provided. (Ann Dermatol (Seoul) 20(3) 130∼133, 2008)

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“…There are different types of collagenomas including familial cutaneous collagenoma [36][37][38] that is characterized by multiple skincolored nodules present on the trunk. The inheritance is autosomal dominant.…”

Section: Fibroblastic Rheumatismmentioning

confidence: 99%

Clinical Features of Scleroderma-Like Disorders: A Challenge for the Rheumatologist

Czirják¹,

Varjú

2006

CRR

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Systemic sclerosis (SSc) is characterized by vascular abnormalities, fibrosis, inflammatory changes, and late stage atrophy/obliterative vasculopathy. Localized scleroderma forms show a longitudinal or circumscribed skin involvement. In scleroderma-like disorders the distribution/characteristics of skin involvement seem to be "atypical" as compared to classic SSc, and the acral skin involvement is usually missing. Exposure to certain chemicals or drugs may also suggest the presence of a scleroderma-like disease. Lack of Raynaud's phenomenon, scleroderma-specific antinuclear antibodies, scleroderma capillary pattern on nailfold capillaroscopy, and typical internal organ manifestations may also indicate the presence of a scleroderma-like disorder. For differential diagnosis skin biopsy is almost always required.Scleroderma-like disorders include diseases with mucin deposition (scleromyxedema, scleredema, etc.). Some disorders show papular-nodular skin changes with or without dermal deposition of materials (amyloid, mucin deposition; fibroblastic rheumatism, etc.). Diseases with monoclonal gammopathy (scleromyxedema, POEMS syndrome, myeloma with scleroderma-like skin changes) also belong to the large group of scleroderma-like diseases. Some disorders are characterized by eosinophilia (diffuse fasciitis with eosinophilia, eosinophilia-myalgia syndrome, toxic oil syndrome), metabolic/biochemical abnormalities (IDDM, nephrogenic fibrosing dermopathy), and endocrine abnormalities (POEMS syndrome, hypo/hyperthyroidism with mucin deposition, diabetes). Chronic graft-versus host disease (cGVHD) may also show scleroderma-like skin changes.Scleroderma-like disorders can be induced by drugs or chemicals (eosinophilia-myalgia syndrome, toxic oil syndrome, vinyl-chloride disease; cytostatic/appetite suppressant, etc), and also by physical injury (trauma, vibration stress, radiation injury). Inherited progeroid syndromes with early ageing (Werner's syndrome, etc.), and a large heterogeneous group of hereditary disorders with either skin thickening (porphyria, phenylketonuria) or skin atrophy/tightening (restrictive dermopathy, scleroatrophic and keratotic dermatosis of the limbs, etc.) should also be taken in consideration in the differential diagnosis of scleroderma-like disorders. These categories are not mutually exclusive, because the remarkably different scleroderma-like diseases show overlapping features.

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Development of collagenomas during pregnancy

Cited by 22 publications

References 11 publications

A Case of Familial Cutaneous Collagenoma

A Case of Familial Cutaneous Collagenoma

A Case of Eruptive Collagenoma on the Left Calf

Clinical Features of Scleroderma-Like Disorders: A Challenge for the Rheumatologist

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