2011
DOI: 10.5897/ajmr10.734
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Determination of the effect of light and salt concentrations on Aphanocapsa algal population

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Cited by 7 publications
(3 citation statements)
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“…The light intensity experiment showed that 6000-7000 Lux was adequate for the algal growth. This observation was in line with [Ifeanyi et al, 2011] who reported that the light with higher intensity can boost the growth of algae. The growth of algae in a reactor can affect the HRAR nutrient removal efficiency [Mirquez et al, 2016], the higher light intensity, the higher of microalgae concentration, resulting in the better removal efficiency.…”
Section: Nitrogen-ammonia Removalsupporting
confidence: 92%
“…The light intensity experiment showed that 6000-7000 Lux was adequate for the algal growth. This observation was in line with [Ifeanyi et al, 2011] who reported that the light with higher intensity can boost the growth of algae. The growth of algae in a reactor can affect the HRAR nutrient removal efficiency [Mirquez et al, 2016], the higher light intensity, the higher of microalgae concentration, resulting in the better removal efficiency.…”
Section: Nitrogen-ammonia Removalsupporting
confidence: 92%
“…Although different algae species require different range of light intensities, most of the algae species show maximum growth in moderately high light intensity. Ifeanyi et al 2011 [ 23 ] experimentally proved a rise in growth rate of Aphanocapsa alga cultured under light of 5000 lux which is about 90 μ molm −2 s −1 .…”
Section: Resultsmentioning
confidence: 99%
“…In response to UV, recent studies showed that mitochondria and chloroplasts produced reactive oxygen species (ROS) that can damage the cellular components (mitochondrial enzymes, membranes) and inhibit cellular metabolic activity (Nawkar et al 2013). According to Yao et al (2004), different stress conditions, such as salinity, temperature, pH, and nutrients (Takagi 2006, Ifeanyi et al 2011, Devi et al 2012, cause changes in mitochondrial transmembrane potential (MTP), releasing cytochrome complex (cyt c) into the intermembrane mitochondrial space, which activates caspase activity and, subsequently, causes cell death. As reported by Mittler et al (2011), ROS can act as secondary signaling molecules, as well as a cell-damaging agent.…”
Section: Discussionmentioning
confidence: 99%