Determination of numbers of lead-exposed American children as a function of lead source: Integrated summary of a report to the U.S. Congress on childhood lead poisoning

Mushak, Paul; Crocetti, Annemarie F.

doi:10.1016/s0013-9351(89)80002-7

Cited by 42 publications

(16 citation statements)

References 12 publications

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“…reported that the young people with dentin lead levels >20 ppm in childhood had a markedly higher risk of dropping out of high school. Increased use of unleaded gasoline and reduction in the lead content of gasoline have been estimated to result in a decrease in mean blood lead levels in the USA (Mushak and Crocetti 1989).…”

Section: Resultsmentioning

confidence: 99%

Lead Levels in Deciduous Teeth of Children from Urban and Suburban Regions in Ankara (Turkey)

Karakaya

İlko

Ulusu

et al. 1996

Bull. Environ. Contam. Toxicol.

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Section: Resultsmentioning

confidence: 99%

Lead Levels in Deciduous Teeth of Children from Urban and Suburban Regions in Ankara (Turkey)

Karakaya

İlko

Ulusu

et al. 1996

Bull. Environ. Contam. Toxicol.

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“…Namely, a noticeable effect of lead on intelligence and on neuropsychological performance in children, which represent a high-risk group, is found (Mushak et al, 1989).…”

Section: Introductionmentioning

confidence: 99%

Distribution of total and organic lead in the Šibenik region (Eastern Adriatic)

Branica

1992

Chemical Speciation & Bioavailability

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The distribution of inorganic and organic lead species in atmospheric and surface waters from the Sibenik area (central part of the Eastern Adriatic coast) was investigated. The concentration range of lead compounds in rainwater was 12,000 to 57,000 ng Pb L-1 for total lead, and 10 to 96 ng Pb L-1 for ionic alkyllead compounds (mostly in the form of trialkyl derivatives). In seawater, trialkyllead compounds were detected in concentration from <0.5 to 12.5 ng Pb-1 L, which represented 0.5 to 6.8% of the total lead. These results were compared with data obtained elsewhere, and the influence of lead compounds emitted from leaded gasoline (from automobiles and gasoline stations) on the adjacent water body is elucidated.

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“…Furthermore, a child's lead exposure history and body burden of lead may strongly influence current PbBs5 and dampen postabatement declines in PbBs. In another study in Baltimore, the mean PbB of a group of lead-poisoned children treated with chelation was below 1.21 ,umol/L upon discharge to lead-free housing but had risen to 1.36 ,umol/L 1 year later. 6 The postdischarge rebound in these children to levels above 1.21 p,mol/L is attributable to high body burdens of lead, not to inadequate abatements. Thus, the decrease in PbBs postabatement may be less than the rise in PbB prevented when a child with a low body burden of lead moves or is bom into a dwelling that has been abated.…”

Section: Outcomes Oflead Pai Abate Ntmentioning

confidence: 92%